Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Laukkanen, M."'
Autor:
Merja Haaparanta-Solin, Maria Domenica Castellone, Juha P. Laurila, Päivi Marjamäki, Mikko O. Laukkanen, Tove J. Grönroos, Lilja E. Laatikainen, Antonio Curcio, Massimo Santoro, Satu Martikainen
Publikováno v:
Molecular Therapy. 17:448-454
Extracellular superoxide dismutase (SOD3) gene therapy has been shown to attenuate tissue damages and to improve the recovery of the tissue injuries, but the cellular events delivering the therapeutic response of the enzyme are not well defined. In t
Autor:
Elina Porkkala-Sarataho, Seppo Ylä-Herttuala, Päivi Turunen, Mikko O. Laukkanen, Pia Leppänen, Jukka T. Salonen
Publikováno v:
Scopus-Elsevier
Clinical and epidemiological studies have provided circumstantial evidence that oxidized low-density lipoprotein (LDL) and antioxidants are involved in the pathogenesis of atherosclerosis. Superoxide dismutases (SODs) have been shown in vitro to prot
Autor:
Massimo Santoro, Lilja E. Laatikainen, Maria Domenica Castellone, Juha P. Laurila, Mikko O. Laukkanen, Mariarosaria Incoronato
Publikováno v:
PLoS ONE
PLoS ONE, Vol 6, Iss 8, p e24456 (2011)
PloS one 6 (2011). doi:10.1371/journal.pone.0024456
info:cnr-pdr/source/autori:Laatikainen, LE [ 1 ] ; Incoronato, M [ 2 ] ; Castellone, MD [ 3 ] ; Laurila, JP [ 1 ] ; Santoro, M [ 3 ] ; Laukkanen, MO [ 1,2]/titolo:SOD3 Decreases Ischemic Injury Derived Apoptosis through Phosphorylation of Erk1%2F2, Akt, and FoxO3a./doi:10.1371%2Fjournal.pone.0024456/rivista:PloS one/anno:2011/pagina_da:/pagina_a:/intervallo_pagine:/volume:6
PLoS ONE, Vol 6, Iss 8, p e24456 (2011)
PloS one 6 (2011). doi:10.1371/journal.pone.0024456
info:cnr-pdr/source/autori:Laatikainen, LE [ 1 ] ; Incoronato, M [ 2 ] ; Castellone, MD [ 3 ] ; Laurila, JP [ 1 ] ; Santoro, M [ 3 ] ; Laukkanen, MO [ 1,2]/titolo:SOD3 Decreases Ischemic Injury Derived Apoptosis through Phosphorylation of Erk1%2F2, Akt, and FoxO3a./doi:10.1371%2Fjournal.pone.0024456/rivista:PloS one/anno:2011/pagina_da:/pagina_a:/intervallo_pagine:/volume:6
BackgroundExtracellular superoxide dismutase (SOD3), which dismutates superoxide anion to hydrogen peroxide, has been shown to reduce the free radical stress derived apoptosis in tissue injuries. Since both superoxide anion and hydrogen peroxide have