Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Jane S Hankins"'
Autor:
Grace Champlin, Winfred C. Wang, Jeremie H. Estepp, Andrew M. Heitzer, Robert F. Davis, Kenneth I. Ataga, Jane S. Hankins, Curtis L. Owens, Guolian Kang, Scott N. Hwang, Allison A. King, Lisa M. Jacola, Juan Ding, Justin Newman
Publikováno v:
Exp Biol Med (Maywood)
Silent cerebral infarcts and arteriopathy are common and progressive in individuals with sickle cell anemia. However, most data describing brain lesions in sickle cell anemia are cross-sectional or derive from pediatric cohorts with short follow-up.
Autor:
Jane S. Hankins, Juan Ding, M. Beth McCarville, Ze Cong, Jeremie H. Estepp, Guolian Kang, Irene Agodoa, Winfred C. Wang
Publikováno v:
British Journal of Haematology. 194:463-468
Children with sickle cell anaemia (SCA) and conditional transcranial Doppler (TCD) flow velocities (conditional: 170-199 cm/s; normal
Autor:
Jason F. Goldberg, Vijaya M. Joshi, Rhiannon R. Penkert, Jane S. Hankins, Amber M Yates, Kenneth I. Ataga, Parul Rai, Victoria I Okhomina, Guolian Kang
Publikováno v:
Blood Adv
Elevated tricuspid regurgitant velocity (TRV) ≥2.5 m/s is a predictor of disease severity in adults and children with sickle cell anemia (SCA), but how disease-modifying therapies (DMTs) affect this biomarker is incompletely understood. We investig
Autor:
Vijaya M. Joshi, Tisha Mentnech, Nicholas S Whipple, Ronak J. Naik, Mary M McFarland, Jane S. Hankins, Vikki G. Nolan
Publikováno v:
Pediatr Blood Cancer
Cardiac disease is the primary cause of death in sickle cell disease (SCD). Cardiac abnormalities begin in childhood and progress throughout life. Right and left ventricular (RV, LV) myocardial strain are early markers of systolic dysfunction but are
Autor:
Ping Zou, Kathleen J. Helton, Jane E. Schreiber, Scott N. Hwang, Jane S. Hankins, Guolian Kang, Juan Ding, Andrew M. Heitzer, Winfred C. Wang
INTRODUCTION Sickle cell anemia (SCA) results in numerous adverse effects on the brain, including neurocognitive dysfunction. Hydroxyurea has been utilized extensively for management of SCA, but its effects on brain function have not been established
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0223fc4d336016fde244b4973c01b871
https://doi.org/10.22541/au.161446690.09460099/v1
https://doi.org/10.22541/au.161446690.09460099/v1
Autor:
Parul Rai, Cara E. Morin, Jane S. Hankins, Akshay Sharma, Amanda Kennedy, Jason N. Johnson, Gary S. Beasley, Richard Lovins, Brandon M. Triplett, Stacy High
Publikováno v:
Blood. 138:3110-3110
Introduction Sickle cell disease (SCD) is characterized by chronic hemolytic anemia which predisposes patients to high output heart failure. Recently it has been recognized that repetitive microvascular ischemic insults and reperfusion injury result
Autor:
Zoltan Patay, Matthew A. Scoggins, Sanjay Verma, Amanda Kennedy, Richard Lovins, Jane S. Hankins, Akshay Sharma, Ping Zou, Ranganatha Sitaram, Brandon M. Triplett, Stacy High
Publikováno v:
Blood. 138:125-125
Introduction Allogeneic hematopoietic cell transplantation (HCT) is increasingly being utilized in patients with sickle cell disease (SCD) to prevent progressive organ dysfunction, including for primary or secondary stroke prevention. However, outcom
Autor:
Mary Beth McCarville, Karen Aldred, Marvin Reid, James M. Boyett, Russell E. Ware, Guolian Kang, Susanna Bortolusso Ali, Jonathan C. Goldsmith, Deanne Soares, Banu Aygun, Matthew P. Smeltzer, Patrícia Moura, Dennis W. Jay, John Bennett, Jane S. Hankins, Clarisse Lopes de Castro Lobo, Angela Rankine-Mullings
Publikováno v:
American Journal of Hematology. 90:1099-1105
Children with sickle cell anemia (SCA) and conditional transcranial Doppler (TCD) ultrasound velocities (170-199 cm/sec) may develop stroke. However, with limited available clinical data, the current standard of care for conditional TCD velocities is
Autor:
Vijaya M. Joshi, Ronak Naik, Guolian Kang, Jason F. Goldberg, Jane S. Hankins, Xiwen Zhao, Kenneth I. Ataga, Parul Rai
Publikováno v:
Blood. 134:1006-1006
Background: Cardiac abnormalities in sickle cell anemia (SCA) include pan-chamber dilation, biventricular hypertrophy, ventricular diastolic dysfunction and elevated pulmonary artery pressures. Diastolic dysfunction in SCA has been reported with a va
Autor:
Winfred C. Wang, Juan Ding, Irene Agodoa, Guolian Kang, Jeremie H. Estepp, Ze Cong, M. Beth McCarville, Jane S. Hankins
Publikováno v:
Blood. 134:1002-1002
Introduction Sickle Cell Anemia (SCA) is a rare, devastating, and debilitating disease marked by the pathophysiologic features of hemolytic anemia, vaso-occlusion, and progressive end-organ damage. The most devastating complication of pediatric SCA i