Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Gavin R. Norton"'
Autor:
Hamza Bello, Ferande Peters, Pinhas Sareli, Adamu J. Bamaiyi, Grace Tade, Angela J. Woodiwiss, Nonhlanhla Mthembu, Suraj M. Yusuf, Patrick H Dessein, Ravi Naran, Carlos D. Libhaber, Vernice R. Peterson, Gavin R. Norton
Publikováno v:
Hypertension (Dallas, Tex. : 1979). 79(2)
Through both backward (Pb) and forward (Pf) wave effects, a lower heart rate (HR) associates with increased central (PPc), beyond brachial pulse pressure (PP). However, the relative contribution to Pf of aortic flow (Q) versus re-reflection of Pb, ha
Autor:
Nonhlanhla Mthembu, Vernice R. Peterson, Hamza Bello, Adamu J. Bamaiyi, Ferande Peters, Angela J. Woodiwiss, Carlos D. Libhaber, Mohlabani Masiu, Keneilwe N. Mmopi, Pinhas Sareli, Gavin R. Norton, Daniel Da Silva Fernandes
Publikováno v:
Hypertension. 75:1574-1583
The relative contribution of loading conditions at different ages across the full adult lifespan to decreases in left ventricular (LV) diastolic function is unclear. Using central arterial pressure and aortic velocity and diameter measurements in the
Autor:
Carlos D. Libhaber, Pinhas Sareli, Vernice R. Peterson, Glenda Norman, Angela J. Woodiwiss, Gavin R. Norton, Monica Gomes
Publikováno v:
BMC Cardiovascular Disorders, Vol 20, Iss 1, Pp 1-9 (2020)
BMC Cardiovascular Disorders
BMC Cardiovascular Disorders
Background Although, in-part through an impact on left ventricular mass (LVM), resistin (an adipokine) may contribute to heart failure, whether this is explained by the adverse effects of resistin on aortic stiffness and renal function is unknown. Me
Publikováno v:
Journal of cardiovascular pharmacology. 79(3)
Myocardial metabolic abnormalities are well-recognized alterations in chronic heart failure, effects that may contribute to progressive cardiac dysfunction. However, whether metabolic alterations in-part mediate their deleterious effects by modifying
Publikováno v:
International Journal of Nephrology and Renovascular Disease
Hon-Chun Hsu,1,2 Chanel Robinson,1 Gavin R Norton,1 Angela J Woodiwiss,1 Patrick H Dessein1,3,4 1Cardiovascular Pathophysiology and Genomics Research Unit, School of Physiology, Faculty of Health Sciences, University of the Witwatersrand, Johannesbur
Autor:
Olebogeng H.I. Majane, Gavin R. Norton, Glenda Norman, Pinhas Sareli, Aletta M.E. Millen, Carlos D. Libhaber, Angela J. Woodiwiss, Frederic S. Michel
Publikováno v:
International Journal of Cardiology. 196:81-87
Although the adipokine resistin may play a role in heart failure, the mechanisms of this effect are uncertain. Relations with left ventricular mass (LVM) and function are uncertain.In 739 randomly selected participants from a community sample (43.6%
Autor:
Sule Gunter, Patrick H Dessein, Linda Tsang, Angela J. Woodiwiss, Frederic S. Michel, Gavin R. Norton, Chanel Robinson, Lebogang Mokotedi, Aletta M.E. Millen
Publikováno v:
Saturday, 16 JUNE 2018.
Background Patients with rheumatoid arthritis (RA) experience an increased frequency of heart failure with a preserved ejection fraction (HFpEF) (1). The treatment of HFpEF is currently suboptimal. Elucidation of the underlying pathophysiological mec
Publikováno v:
American journal of hypertension. 31(9)
BACKGROUND Blunted nocturnal blood pressure (BP) dipping (nondipping) predicts cardiovascular morbidity and mortality, and is associated with heart failure (HF) independent of office BP. Whether nondipping is independently associated with cardiac sys
Autor:
Patrick H Dessein, Aletta M.E. Millen, Chanel Robinson, Linda Tsang, Angela J. Woodiwiss, Sule Gunter, Gavin R. Norton, Lebogang Mokotedi
Publikováno v:
Poster Presentations.
Background Heart failure contributes to the excess mortality experienced by patients with rheumatoid arthritis (RA) (1). Impaired diastolic function represents a pre-clinical cardiac alteration which is highly predictive of cardiac events and often p
Publikováno v:
American journal of hypertension. 31(2)
BACKGROUND Left ventricular (LV) diastolic dysfunction characterizes heart failure with a preserved ejection fraction. Although it is recognized that the renin–angiotensin–aldosterone system (RAAS) decreases LV diastolic function, whether systemi