Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Michal Goldberg"'
Autor:
Michael Milyavsky, Lital Eini, Shlomit Ezer, Eran Meshorer, Ayala Gold, Rotem Hanania, Michal Goldberg, Ruth Viner, Malka Nissim-Rafinia, Keren Erez, Nasma Aqaqe
Publikováno v:
Oncogene. 38:3103-3118
The cancer stem cell (CSC) model suggests that a subpopulation of cells within the tumor, the CSCs, is responsible for cancer relapse and metastasis formation. CSCs hold unique characteristics, such as self-renewal, differentiation abilities, and res
Autor:
Jardena Nordenberg, Steven Henick, Hadar Goldvaser, Einat Beery, Anna Gutkin, Orit Uziel, Meir Lahav, Michal Goldberg, Maria Pinchasi
Publikováno v:
Oncotarget
Exosomes are small (30-100nm) vesicles secreted from all cell types serving as inter-cell communicators and affecting biological processes in “recipient” cells upon their uptake. The current study demonstrates for the first time that hTERT mRNA,
Publikováno v:
Oncogene. 31:3495-3504
DNA double-strand breaks (DSBs), the most hazardous DNA lesions, may result in genomic instability, a hallmark of cancer cells. The main DSB repair pathways are non-homologous end joining (NHEJ) and homologous recombination (HR). In mammalian cells,
Autor:
Diane Genet, Bernard S. Lopez, Alkmini Kalousi, B. Fischer, Michal Goldberg, P Betrand, C. Lemaître, Josée Guirouilh-Barbat, A-S Hoffbeck, Or David Shahar, Evi Soutoglou, Laurent Brino
Publikováno v:
Oncogene
Oncogene, 2012, 31 (45), pp.4803-4809. ⟨10.1038/onc.2011.638⟩
Oncogene, Nature Publishing Group, 2012, 31 (45), pp.4803-4809. ⟨10.1038/onc.2011.638⟩
Oncogene, 2012, 31 (45), pp.4803-4809. ⟨10.1038/onc.2011.638⟩
Oncogene, Nature Publishing Group, 2012, 31 (45), pp.4803-4809. ⟨10.1038/onc.2011.638⟩
International audience; DNA repair is essential in maintaining genome integrity and defects in different steps of the process have been linked to cancer and aging. It is a long lasting question how DNA repair is spatially and temporarily organized in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d3aa9348c8085b050da102079336a161
https://hal.science/hal-03048142
https://hal.science/hal-03048142
Autor:
Yoav Smith, Ephrat Levy-Lahad, Lital Eini, Michal Goldberg, Amnon Lahad, Dana Bashari, Inbal Gazy, David A. Zeevi, Sharon Zeligson, Paul Renbaum, Doron Ginsberg
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 7, p e0134120 (2015)
PLoS ONE, Vol 10, Iss 7, p e0134120 (2015)
Expression of RAD51, a crucial player in homologous recombination (HR) and DNA double-strand break (DSB) repair, is dysregulated in human tumors, and can contribute to genomic instability and tumor progression. To further understand RAD51 regulation