Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Bingjie Lv"'
Interleukin 35 ameliorates myocardial ischemia‐reperfusion injury by activating the gp130‐STAT3 axis
Autor:
Bingjie Lv, Jiao Jiao, Chanjuan Xu, Guofei Hou, Xiang Cheng, Xiang-Ping Yang, Ni Xia, Yu Hu, Shaofang Nie, Jianfeng Liu, Min Zhang, Tingting Tang, Yuhua Liao, Xingdi Zhou
Publikováno v:
The FASEB Journal. 34:3224-3238
Myocardial ischemia-reperfusion injury (MIRI) is common clinical complication, which represents significant challenge in the treatment of acute myocardial infarction (AMI) diseases. Interleukin 35 (IL-35) exhibits anti-inflammatory properties via the
Autor:
Muyang Gu, Guo-Ping Shi, Yuzhi Lu, Jingyong Li, Jiao Jiao, Ni Xia, Xiang-Ping Yang, Bingjie Lv, Shaofang Nie, Yu Hu, Dan Li, Shuang Wen, Tingting Tang, Xiang Cheng, Mengyang Liao, Yuhua Liao
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 39:446-458
Objective— Inflammation occurs during the progression of abdominal aortic aneurysm (AAA). IL (interleukin)-33 is a pleiotropic cytokine with multiple immunomodulatory effects, yet its role in AAA remains unknown. Approach and Results— Immunoblot,
Autor:
Min Zhang, Ting Zhou, Meilin Liu, Ni Xia, Muyang Gu, Tingting Tang, Shaofang Nie, Zhengfeng Zhu, Bingjie Lv, Jiao Jiao, Xiangping Yang, Xiang Cheng
Publikováno v:
Biochemical and biophysical research communications. 547
Fibrotic scarring is tightly linked to the development of heart failure in patients with post-myocardial infarction (MI). Atypical chemokine receptor 4 (ACKR4) can eliminate chemokines, such as C–C chemokine ligand 21 (CCL21), which is independentl
Autor:
Yuzhi Lu, Tongjie Xu, Yuhua Liao, Shaofang Nie, Shuang Wen, Ni Xia, Bing Sun, Bingjie Lv, Chenping Jia, Wenyong Dong, Daan Nie, Yuan Yuan Li, Jiao Jiao, Longxian Cheng, Tingting Tang, Zhengfeng Zhu, Xiang Cheng, Wen-cai Zhang, Su-Feng Zhou
Publikováno v:
Cardiovascular research. 106(3)
Aims Recently, interleukin (IL)-9 was found to be involved in the pathogenesis of many inflammatory diseases. Here, we tested whether IL-9 was related to atherosclerosis and investigated the underlying mechanisms. Methods and results IL-9R was expres