Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Satoshi Mano"'
Publikováno v:
Intractable Rare Dis Res
Hereditary angioedema due to C1-inhibitor (C1-INH) deficiency (HAE-C1-INH) induces an acute attack of angioedema. In 2018, icatibant available for self-possession and subcutaneous self-administration was licensed for on-demand treatment in addition t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c2e6d7201153647ab45673a8ef35368
https://europepmc.org/articles/PMC8122312/
https://europepmc.org/articles/PMC8122312/
Autor:
Hiroyuki Inoshita, Isao Ohsawa, Kisara Onda, Nobuyuki Sato, Gaku Kusaba, Masaya Ishii, Hiroyuki Ohi, Satoshi Mano, Yasuhiko Tomino, Tomohito Gohda, Satoshi Horikoshi
Publikováno v:
Journal of Clinical Laboratory Analysis. 24:113-118
The aim of this study was to explore the association between the serum concentration of complement component 3 (C3) and a variety of metabolic parameters. The study involved 125 patients in our outpatient clinic. Anthropometric and clinical laborator
Autor:
Nobuyuki Sato, Satoshi Horikoshi, Yasuhiko Tomino, Isao Ohsawa, Hiroyuki Inoshita, Satoshi Mano, Daisuke Honda
Publikováno v:
Immunobiology. 221:1136
Autor:
Kiyoshi Matsuoka, Yukihiko Takeda, Miyuki Akiba-Takagi, Isao Ohsawa, Satoshi Horikoshi, Shinya Ogasawara, Masanori Hara, Yasuhiko Tomino, Rin Asao, Yoshiko Nagai-Hosoe, Satoshi Mano, Yoshiaki Hirayama, Takuto Seki, Fumiko Kodama, Sakari Sekine, Katsuhiko Asanuma, Hiroyuki Kurosawa
Publikováno v:
Clinical journal of the American Society of Nephrology : CJASN. 7(9)
Podocalyxin (PCX) is present on the apical cell membrane of podocytes and is shed in urine from injured podocytes. Urinary podocalyxin (u-PCX) is associated with severity of active glomerular injury in patients with glomerular diseases. This study ex
Autor:
Teizo Fujita, Isao Ohsawa, Mariko Tamano, Hiroyuki Ohi, Yasuhiko Tomino, Satoshi Horikoshi, Kisara Onda, Akie Toki, Satoshi Mano, Michiro Wakabayashi
Publikováno v:
BMC Nephrology
BMC Nephrology, Vol 12, Iss 1, p 64 (2011)
BMC Nephrology, Vol 12, Iss 1, p 64 (2011)
Background Glomerular damage in IgA nephropathy (IgAN) is mediated by complement activation via the alternative and lectin pathways. Therefore, we focused on molecules stabilizing and regulating the alternative pathway C3 convertase in urine which mi