Zobrazeno 1 - 10
of 105
pro vyhledávání: '"Yunzeng Zou"'
Autor:
Jian Wu, Cheng Yang, Jun Ren, Xiangdong Yang, Hiroshi Akazawa, Guoping Zhang, Chunjie Yang, Komuro Issei, Yunzeng Zou, Ning Zhou, Le Kang, Li Lin, Qi Wang, Xiaoyan Wang, Yunqin Chen, Ruizhen Chen, Junbo Ge, Hong Jiang, Aijun Sun, Hui Gong
Publikováno v:
Journal of Molecular and Cellular Cardiology. 162:110-118
It is well known that lectin-like oxidized low-density lipoprotein (ox-LDL) and its receptor LOX-1, angiotensin II (AngII) and its type 1 receptor (AT1-R) play an important role in the development of cardiac hypertrophy. However, the molecular mechan
Autor:
Xiangfei Wang, Suling Ding, Yunzeng Zou, Xiaowei Zhu, Xiangdong Yang, Jian Wu, Weiwei Zhang, Baoling Zhu, Junbo Ge
Publikováno v:
Journal of Cardiovascular Translational Research. 15:621-634
Myocardial infarction (MI) is a significant contributor to the development of heart failure. Histidine decarboxylase (HDC), the unique enzyme that converts l-histidine to histamine, is highly expressed in CD11b+ immature myeloid cells. However, the r
Autor:
Junbo Ge, Minghui Li, Xiaoxiao Liu, Yucheng Wang, Yong Yu, Yunzeng Zou, Ying Yu, Ruizhen Chen, Hui Shi
Publikováno v:
Cell Biology and Toxicology. 38:487-504
Cardiomyocyte apoptosis is critical for the development of viral myocarditis (VMC), which is one of the leading causes of cardiac sudden death in young adults. Our previous studies have demonstrated that elevated calpain activity is involved in the p
Autor:
Yong Yu, Junbo Ge, Xiaoxiao Liu, Minghui Li, Yunzeng Zou, Yucheng Wang, Hui Shi, Ying Yu, Ruizhen Chen
Publikováno v:
International Heart Journal. 62:900-909
Virus myocarditis (VMC) is a common cardiovascular disease and a major cause of sudden death in young adults. However, there is still a lack of effective treatments. Our previous studies found that calpain activation was involved in VMC pathogenesis.
Autor:
Yunzeng Zou, Lei Zhang, Jingfeng Wang, Baoli Zhang, Jian Wu, Ying Yu, Junbo Ge, Hong Jiang, Yangang Su
Publikováno v:
International Heart Journal. 62:162-170
High-mobility group box 1 (HMGB1) is increased in the myocardium under pressure overload (PO) and is involved in PO-induced cardiac remodeling. The mechanisms of the upregulation of cardiac HMGB1 expression have not been fully elucidated. In the pres
Autor:
Aijun Sun, Hang Chen, Hao Jiang, Heng Yang, Yongchao Zhao, Yunzeng Zou, Xiaolei Sun, Jin Liu, Wenjia Li, Jingjing Hu, Rifeng Gao, Zhen Dong, Junbo Ge
Publikováno v:
Bioactive Materials
Bioactive Materials, Vol 6, Iss 7, Pp 2058-2069 (2021)
Bioactive Materials, Vol 6, Iss 7, Pp 2058-2069 (2021)
Mitochondrial damage is a critical driver in myocardial ischemia-reperfusion (I/R) injury and can be alleviated via the mitochondrial transplantation. The efficiency of mitochondrial transplantation is determined by mitochondrial vitality. Because al
Publikováno v:
Journal of Cellular and Molecular Medicine
To investigate the prevention of cardiac remodelling and inflammatory immune response after myocardial infarction (MI) via ACEI regulating dendritic cells (DCs), we explored whether the protective effect of ACEI was repressed under hyperlipidemic env
Autor:
Jingfeng Wang, Yanyan Wang, Jingmin Zhou, Xiaotong Cui, Junbo Ge, Juan Cao, Xueting Han, Aijun Sun, Kai Hu, Yunzeng Zou, Jie Luo, Mingqiang Fu, Yuyuan Fan, Yu Song
Publikováno v:
Journal of Cardiovascular Translational Research
Diastolic dysfunction is common in various cardiovascular diseases, which could be affected by adiponectin (APN). Nevertheless, the effects of APN on diastolic dysfunction in pressure overload model induced by transverse aorta constriction (TAC) rema
Autor:
Fangjie Dai, Yunzeng Zou, Xuan Li, Peipei Yin, Xia Li, Shijun Wang, Junbo Ge, Zhiwen Ding, Jian Wu, Ran Xu
Publikováno v:
Frontiers in Molecular Biosciences, Vol 8 (2021)
Frontiers in Molecular Biosciences
Frontiers in Molecular Biosciences
Cardiac hypertrophic preconditioning (HP) signifies cardioprotection induced by transient pressure overload to resist hypertrophic effects of subsequently sustained pressure overload. Although it is recently found that inflammation triggers the devel
Autor:
Yangang Su, Ai-jun Sun, Yunzeng Zou, Hong Jiang, Lei Zhang, Guoliang Jiang, Ying Yu, Xue Yang, Junbo Ge, Jian Wu
Publikováno v:
International Journal of Molecular Medicine
Pressure overload‑induced cardiac hypertrophy is associated with a complex spectrum of pathophysiological mechanisms, including the inflammation response. High mobility group box‑1 (HMGB1), a pro‑inflammatory cytokine, is not only increased in