Zobrazeno 1 - 10
of 14
pro vyhledávání: '"Xiao-Qian Li"'
Autor:
He Huang, Li Min Chen, Xiao Jie Fang, Cheng Cheng Guo, Xiao Ping Lin, Huang Ming Hong, Xi Li, Zhao Wang, Ying Tian, Mei Ting Chen, Yu Yi Yao, Zegeng Chen, Xiao Qian Li, Fei Pan
Publikováno v:
Frontiers in Pharmacology, Vol 11 (2020)
Frontiers in Pharmacology
Frontiers in Pharmacology
BackgroundExtranodal natural killer/T-cell lymphoma (ENKTL) is a rare and extremely malignant tumor. The systemic inflammation score (SIS), which is based on the pretreatment level of lymphocyte-to-monocyte ratio (LMR) and serum albumin (Alb), has b
Publikováno v:
Nature and Science of Sleep
Yu-wei Zhang,1,2 Xiao-qian Li,1 Wen-fei Tan,1 Bo Fang,1 Hong Ma1 1Department of Anesthesiology, The First Hospital of China Medical University, Shenyang, People’s Republic of China; 2Department of Anesthesiology, The First Hospital of Hebei Medical
Publikováno v:
Oxidative Medicine and Cellular Longevity
Oxidative Medicine and Cellular Longevity, Vol 2020 (2020)
Oxidative Medicine and Cellular Longevity, Vol 2020 (2020)
Background. Neuron survival after ischemia-reperfusion (IR) injury is the primary determinant of motor function prognosis. MicroRNA- (miR-) based gene therapy has gained attention recently. Our previous work explored the mechanisms by which miR-137-3
Autor:
Jun Wang, Yanhua Jiang, Hong Ma, Fang Liu, Xuezhao Cao, Xiao-Qian Li, Xi-Jia Sun, Zhe Li, Ayong Tian
Publikováno v:
Cellular Physiology and Biochemistry, Vol 46, Iss 4, Pp 1398-1411 (2018)
Background/Aims: TREM2 plays a crucial role in modulating microglial function through interaction with DAP12, the adapter for TREM2. Emerging evidence has demonstrated that TREM2 could suppress neuroinflammatory responses by repression of microglia-m
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-12 (2017)
Journal of Neuroinflammation
Journal of Neuroinflammation
Background Ischemia-reperfusion (IR) affects microRNA (miR) expression and causes substantial inflammation. Multiple roles of the tumor suppressor miR-129-5p in cerebral IR have recently been reported, but its functions in the spinal cord are unclear
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-13 (2018)
Journal of Neuroinflammation
Journal of Neuroinflammation
Background Ischaemia reperfusion (IR) induces multiple pathophysiological changes. In addition to its classical role in regulating tumourigenesis, the feedback loop formed by p53 and its driven target p53-upregulated modulator of apoptosis (PUMA) was
Autor:
Wen-Fei Tan, Hong Ma, Ying Zhang, Xiao-Li Pi, Xiao-Qian Li, Bo Fang, Na-Ren Bao, Fengshou Chen
Publikováno v:
Neuroscience. 328:107-116
Autophagy plays an important role in spinal cord ischemia reperfusion (I/R) injury, but its neuroprotective or neurodegenerative role remains controversial. The extent and persistence of autophagy activation may be the critical factor to explain the
Publikováno v:
Brain Research. 1727:146489
Spinal cord ischemia-reperfusion injury (SCII) remains an unresolved complication and its underlying mechanism has not been fully elucidated. In this study, we studied the role of chemokine (C-X-C motif) ligand 13 (CXCL13) in a rat model of SCII. We
Publikováno v:
BMJ Open Diabetes Research & Care
ObjectiveDiabetic nephropathy (DN) is the leading cause of chronic kidney disease and end-stage renal disease. Emerging evidence suggests that complement activation is involved in the pathogenesis of DN. The aim of this study was to investigate the p
Publikováno v:
PLoS ONE
PLoS ONE, Vol 11, Iss 10, p e0163807 (2016)
PLoS ONE, Vol 11, Iss 10, p e0163807 (2016)
Toll-like receptor 4 (TLR4) is important for the pathogenesis of inflammatory reactions and the promotion of pain processing after ischemia/reperfusion (IR) in spinal cord. Recently, C-X-C chemokine ligand 12 (CXCL12) and its receptor, C-X-C chemokin