Zobrazeno 1 - 10
of 1 284
pro vyhledávání: '"11"'
Publikováno v:
Archives of pathologylaboratory medicine. 145(8)
Trophoblast destruction may act as a potential mechanism to allow the virus to penetrate the chorionic villi and, once it has reached the fetal vessels, to become widespread in the fetal circulation, persisting in fetal vessels and macrophages, as sh
Publikováno v:
Prenatal Diagnosis. 25:683-686
Objective To report the first tertiary monosomy in a pregnancy loss to a female t(11;22) carrier. Methods The patient was a 34-year-old G10P1 female known to have a balanced translocation t(11;22)(q23;q11.2). She had one female livebirth (a transloca
Publikováno v:
Journal of developmental origins of health and disease. 8(2)
Maternal physiologic stress during gestation has been reported to be associated with negative developmental outcomes, including intra-uterine growth restriction and reduced birth weight, which can impact postnatal development, behavior and health. Th
Autor:
Licia Turolla, Licia Laurino, Demetrio Baldo, Nicola Surico, Renzo Boldorini, Lidia Larizza, Giuseppe Simoni, Patrizia D’Ajello, F.R. Grati, Anna Maria Ruggeri, Monica Miozzo, Gabriella Bracalente, Federico Maggi, Elisabetta Frate
Introduction: The phenotypic variability in Beckwith-Wiedemann Syndrome reflects the genetic heterogeneity of the mechanisms which default leads to deregulation of genes located at 11p15.5. Genotype-phenotype correlation studies demonstrated an assoc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e3b2663f31bef892c439537e1cd4e8a6
http://hdl.handle.net/2434/42144
http://hdl.handle.net/2434/42144
Autor:
Silvio Zaina, Gloria Barbosa-Sabanero, Martha-Olivia Solís-Martínez, Katarzyna Wrobel, Gustavo Romero-Gutiérrez, Maria-Luisa Lazo-de-la-Vega-Monroy, Kazimierz Wrobel, Victor E. Aguirre-Arzola
Publikováno v:
Steroids. 124
Small for gestational age infants have greater risk of developing metabolic diseases in adult life. It has been suggested that low birth weight may result from glucocorticoid excess in utero, a key mechanism in fetal programming. The placental enzyme
Publikováno v:
Prenatal diagnosis. 11(7)
Fetal chromosome analysis in a 39-year-old mother revealed a chromosome 11 aberration interj ireted as a duplication of the centromere. This was also found in the mother's karyotype, raising the possibility that the abnormality was a new variant of n
Autor:
Kaiping Yang
Publikováno v:
Reviews of Reproduction. 2:129-132
During mammalian pregnancy, the circulating concentration of cortisol (in rodents, corticosterone) in the mother is much higher than that in the fetus. Since the placenta is the only barrier, apart from the uterus, between the mother and her fetus, t
Publikováno v:
Genetical Research. 68:35-43
SummaryPrevious studies have shown that mice with paternal disomy for chromosome 11 are consistently larger at birth than their normal sibs, whereas mice with the maternal disomy are consistently smaller. An imprinting effect with monoallelic express
Autor:
Robbie S. Lindsay, Caroline Mckenzie Leckie, Parvez Murad, John J. Mullins, Christopher R. W. Edwards, Yuri Kotelevtsev, Jonathan R. Seckl, Roger W. Brown, Lawrence P. Brett, Karen E. Chapman, Joyce L.W. Yau, Val Lyons
Publikováno v:
Biochemical Journal. 313:1007-1017
By inactivating potent glucocorticoid hormones (cortisol and corticosterone), 11 beta-hydroxysteroid dehydrogenase type 2 (11 beta-HSD2) plays an important role in the placenta by controlling fetal exposure to maternal glucocorticoids, and in aldoste
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 80:885-890
Two isoforms of 11 beta-hydroxysteroid dehydrogenase (11 beta HSD) have been described which catalyze the interconversion of cortisol (F) to cortisone (E). 11 beta HSD activity has previously been reported in placenta and fetal membranes, where its r