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of 4
pro vyhledávání: '"Morten Dahl"'
Autor:
Lars Christian Laursen, Sadaf Qayum, Morten Dahl, Ronald Dahl, Pierre Bouchelouche, Anne Orholm Nielsen
Publikováno v:
Nielsen, A O, Qayum, S, Bouchelouche, P N, Laursen, L C, Dahl, R & Dahl, M 2016, ' Risk of asthma in heterozygous carriers for cystic fibrosis : A meta-analysis ', Journal of Cystic Fibrosis, vol. 15, no. 5, pp. 563–567 . https://doi.org/10.1016/j.jcf.2016.06.001
Background: The high prevalence of asthma in patients with cystic fibrosis (CF) has been known for long. However, it is still unclear whether heterozygous CF carriers are susceptible to asthma. Given this, a meta-analysis is necessary to determine th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3aee55ea70dff18f5cae77fb0e728352
https://findresearcher.sdu.dk:8443/ws/files/123311614/Risk_of_asthma_in_heterozygous_carriers_for_cystic_fibrosis.pdf
https://findresearcher.sdu.dk:8443/ws/files/123311614/Risk_of_asthma_in_heterozygous_carriers_for_cystic_fibrosis.pdf
Autor:
Børge G. Nordestgaard, Jonathan Lam, Stewart J. Levine, Maryann Kaler, Xianglan Yao, Jing-Ping Lin, Meixia Gao, Gyorgy Csako, Xuan Qu, Robert D. Shamburek, Zu-Xi Yu, Cuilian Dai, Neal Jeffries, Morten Dahl, Alan T. Remaley, Gayle Z. Nugent, Karin Fredriksson, Karen J. Keeran, Rene Costello
Publikováno v:
American journal of physiology. Lung cellular and molecular physiology. 302(2)
Apolipoprotein E (apoE) is an endogenous negative regulator of airway hyperreactivity (AHR) and mucous cell metaplasia in experimental models of house dust mite (HDM)-induced airway disease. The gene encoding human apoE is polymorphic, with three com
Autor:
Thomas J. Mariani, Robert H. Lim, Lester Kobzik, Adriana S. Leme, Alexey V. Fedulov, Zhiping Yang, Morten Dahl
Publikováno v:
American journal of respiratory cell and molecular biology. 38(1)
Maternal immune responses can promote allergy development in offspring, as shown in a model of increased susceptibility to asthma in babies of ovalbumin (OVA)-sensitized and -challenged mother mice. We investigated whether inflammatory responses to a
Publikováno v:
Respiratory Medicine. (9):1286-1292
SummaryThe angiotensin-converting enzyme (ACE) gene is a potential candidate gene for risk of asthma, COPD, and COPD co-morbidity. In 9034 Danish adults, we determined whether individuals homozygous or heterozygous for the ACE D allele are at greater