Zobrazeno 1 - 10
of 267
pro vyhledávání: '"76"'
Autor:
Craig M. Williams, Jenny P. Johns, Achim Porzelle, Paul Reddell, Heiko Schill, Lin Dong, Peter G. Parsons, Rebecca L. Grange, Victoria A. Gordon
Publikováno v:
Chemistry (Weinheim an der Bergstrasse, Germany). 15(42)
EBC-23, 24, 25, 72, 73, 75 and 76 were isolated from the fruit of Cinnamomum laubatii (family Lauraceae) in the Australian tropical rainforests. EBC-23 (1) was synthesized stereoselectively, in nine linear steps in 8 % overall yield, to confirm the r
Autor:
Jack D. Keene, Elena Y. Dobrikova, Valentina Neplioueva, Neelanjan Mukherjee, Matthias Gromeier
Publikováno v:
PLoS ONE, Vol 5, Iss 7, p e11710 (2010)
PLoS ONE
PLoS ONE
Background RNA-binding proteins accompany all steps in the life of mRNAs and provide dynamic gene regulatory functions for rapid adjustment to changing extra- or intracellular conditions. The association of RNA-binding proteins with their targets is
Autor:
K P Kushwaha, Akhilesh C. Mishra, Shobha D. Chitambar, Pradip V. Fulmali, Vijay P. Bondre, Pooja R. Patil, Varanasi Gopalkrishna, Daya Gangale, Vijay M. Ayachit, Gajanan N. Sapkal, Milind M. Gore, A K Rathi, Vipul Dadhania
Publikováno v:
Emerging Infectious Diseases
Emerging Infectious Diseases, Vol 15, Iss 2, Pp 295-298 (2009)
Emerging Infectious Diseases, Vol 15, Iss 2, Pp 295-298 (2009)
An outbreak of viral encephalitis occurred in northern India in 2006. Attempts to identify an etiologic agent in cerebrospinal fluid by using reverse transcription–PCR showed positivity to enterovirus (EV) in 66 (21.6%) of 306 patients. Sequencing
Autor:
Jingwen Feng, Songlin Li, Bing Zhang, Namin Duan, Rui Zhou, Shike Yan, Jeevithan Elango, Ning Liu, Wenhui Wu
Publikováno v:
Marine Drugs, Vol 20, Iss 76, p 76 (2022)
Marine Drugs
Marine Drugs; Volume 20; Issue 1; Pages: 76
Marine Drugs
Marine Drugs; Volume 20; Issue 1; Pages: 76
FGFC1, an active compound isolated from the culture of marine fungi Stachybotrys longispora FG216, elicits fibrinolytic, anti-oxidative, and anti-inflammatory activity. We have previously reported that FGFC1 inhibited the proliferation, migration, an
Autor:
Céline Delierneux, Brett G. Hollier, Justine Lambert, Marc Thiry, Erik W. Thompson, Agnès Noël, Silvia Blacher, Meggy Suarez-Carmona, Guy Jerusalem, Morgane Bourcy, Myriam Polette, Hélène Schroeder, Marie-Emilie Francart, Geert Berx, Nicolas Skrypek, Cécile Oury, Christine Gilles
Publikováno v:
Cancer Research
Cancer Research, American Association for Cancer Research, 2016, 76 (14), pp.4270-4282. ⟨10.1158/0008-5472.CAN-15-2263⟩
Cancer Research, 2016, 76 (14), pp.4270-4282. ⟨10.1158/0008-5472.CAN-15-2263⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (14), pp.4270-4282. ⟨10.1158/0008-5472.CAN-15-2263⟩
Cancer Research, 2016, 76 (14), pp.4270-4282. ⟨10.1158/0008-5472.CAN-15-2263⟩
Epithelial–mesenchymal transition (EMT) is prominent in circulating tumor cells (CTC), but how it influences metastatic spread in this setting is obscure. Insofar as blood provides a specific microenvironment for tumor cells, we explored a potentia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f38143fe725257e80b0e090888f2bee0
https://hal.univ-reims.fr/hal-02448692
https://hal.univ-reims.fr/hal-02448692
Autor:
Imade Ait-Arsa, Leïla Houhou, Frédéric Hollande, Charles Vincent, Julie Giraud, Laura M. Failla, Julie Pannequin, André Pèlegrin, Jean-François Bourgaux, Imène Gasmi, Daniel Birnbaum, Jean-Marc Pascussi, Jérémy Ollier, Thibault Mazard, Michel Prudhomme, James G. Ryall, Pascal Finetti, François Bertucci, Chu Ya, Dominique Joubert, Ebba Louise Lagerqvist
Publikováno v:
Cancer Research
Cancer Research, 2016, 76 (12), pp.3618-3628. ⟨10.1158/0008-5472.CAN-15-1497⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (12), pp.3618-3628. ⟨10.1158/0008-5472.CAN-15-1497⟩
Cancer Research, 2016, 76 (12), pp.3618-3628. ⟨10.1158/0008-5472.CAN-15-1497⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (12), pp.3618-3628. ⟨10.1158/0008-5472.CAN-15-1497⟩
Subpopulations of cancer stem–like cells (CSC) are thought to drive tumor progression and posttreatment recurrence in multiple solid tumors. However, the mechanisms that maintain stable proportions of self-renewing CSC within heterogeneous tumors u
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::999921ab65c1e80a5202eb28b7425eba
https://hal.science/hal-01905320
https://hal.science/hal-01905320
Autor:
Qi Cao, Brendan Pang, Erkan Baloglu, Zhiqiang Zhao, Helene Marijon, Huey Jin Lim, Ngan B. Doan, Xiao Guo, Haibo Sun, Charles Forscher, De-Chen Lin, Henry Yang, Raja Rajalingam, H. Phillip Koeffler, Sharon Shacham, Sigal Gery, Victor Kwan Min Lee, Peiguo Chu, Anand Mayakonda, Tom Thomas, Jonathan W. Said, Liang Xu
Publikováno v:
Cancer research, vol 76, iss 9
Sun, H; Lin, DC; Cao, Q; Guo, X; Marijon, H; Zhao, Z; et al.(2016). CRM1 inhibition promotes cytotoxicity in ewing sarcoma cells by repressing EWS-FLI1-dependent IGF-1 signaling. Cancer Research, 76(9), 2687-2697. doi: 10.1158/0008-5472.CAN-15-1572. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/1x97j4wp
Sun, H; Lin, DC; Cao, Q; Guo, X; Marijon, H; Zhao, Z; et al.(2016). CRM1 inhibition promotes cytotoxicity in ewing sarcoma cells by repressing EWS-FLI1-dependent IGF-1 signaling. Cancer Research, 76(9), 2687-2697. doi: 10.1158/0008-5472.CAN-15-1572. UC San Francisco: Retrieved from: http://www.escholarship.org/uc/item/1x97j4wp
Ewing sarcoma (EWS) is an aggressive bone malignancy that mainly affects children and young adults. The mechanisms by which EWS (EWSR1) fusion genes drive the disease are not fully understood. CRM1 (XPO1) traffics proteins from the nucleus, including
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d8a286a62eeb29fdb6fab5d0ca26c6e0
https://escholarship.org/uc/item/1x97j4wp
https://escholarship.org/uc/item/1x97j4wp
Autor:
Sebastien Benzekry, John M.L. Ebos, Dominique Barbolosi, Ryan Corbelli, Michalis Mastri, Amanda Tracz
Publikováno v:
Cancer Research
Cancer Research, 2016, 76 (3), pp.535-547. ⟨10.1158/0008-5472.CAN-15-1389⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (3), pp.535-547. ⟨10.1158/0008-5472.CAN-15-1389⟩
Cancer Research, 2016, 76 (3), pp.535-547. ⟨10.1158/0008-5472.CAN-15-1389⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (3), pp.535-547. ⟨10.1158/0008-5472.CAN-15-1389⟩
Rapid improvements in the detection and tracking of early-stage tumor progression aim to guide decisions regarding cancer treatments as well as predict metastatic recurrence in patients following surgery. Mathematical models may have the potential to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9823ab3c0a8f14352d208ff5a55c9452
https://inria.hal.science/hal-01222046
https://inria.hal.science/hal-01222046
Autor:
Lise Queiroz, Francis Bonnefoy, Béatrice Gaugler, Clémentine Gamonet, Sindy Vrecko, Olivier Adotevi, Eric Tartour, Antoine Thiery-Vuillemin, Thierry Nguyen Tan Hon, Jean-René Pallandre, Laura Mansi, Christophe Borg, Guillaume Mouillet, Patrice Ravel, Elsa Curtit, Yann Godet, Tristan Maurina, Jagadeesh Bayry, Caroline Laheurte, Elodie Lauret Marie-Joseph, Laura Boullerot, Bernard Royer, Xavier Pivot, Laurent Beziaud, L. Rangan
Publikováno v:
Cancer Research
Cancer Research, 2016, 76 (14), pp.4100-4112. ⟨10.1158/0008-5472.CAN-15-2452⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (14), pp.4100-4112. ⟨10.1158/0008-5472.CAN-15-2452⟩
Cancer Research, 2016, 76 (14), pp.4100-4112. ⟨10.1158/0008-5472.CAN-15-2452⟩
Cancer Research, American Association for Cancer Research, 2016, 76 (14), pp.4100-4112. ⟨10.1158/0008-5472.CAN-15-2452⟩
The rapalogs everolimus and temsirolimus that inhibit mTOR signaling are used as antiproliferative drugs in several cancers. Here we investigated the influence of rapalogs-mediated immune modulation on their antitumor efficacy. Studies in metastatic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::02f2fa176a5697c8523397639af997f1
https://hal.umontpellier.fr/hal-02294356
https://hal.umontpellier.fr/hal-02294356
Autor:
Prunier, Chloé, Josserand, Véronique, Vollaire, Julien, Beerling, Evelyne, Koning, Leanne, Petropoulos, Christos, Destaing, Olivier, Hurbin, Amandine, Montemagno, Christopher, Prudent, Renaud, De Koning, Leanne, Kapur, Reuben, Cohen, Pascale A., Albiges-Rizo, Corinne, Coll, Jean-Luc, Rheenen, Jacco, Billaud, Marc, Lafanechere, Laurence
Publikováno v:
Cancer Research
Cancer Research, 76(12), 3541-3552
Cancer Research, 76(12), 3541-52. American Association for Cancer Research Inc.
Cancer Research, American Association for Cancer Research, 2016, 76 (12), pp.3541-3552. ⟨10.1158/0008-5472.CAN-15-1864⟩
Cancer Research, 76(12), 3541. American Association for Cancer Research Inc.
Cancer Research, 2016, 76 (12), pp.3541-3552. ⟨10.1158/0008-5472.CAN-15-1864⟩
Cancer Research, 76(12), 3541-3552
Cancer Research, 76(12), 3541-52. American Association for Cancer Research Inc.
Cancer Research, American Association for Cancer Research, 2016, 76 (12), pp.3541-3552. ⟨10.1158/0008-5472.CAN-15-1864⟩
Cancer Research, 76(12), 3541. American Association for Cancer Research Inc.
Cancer Research, 2016, 76 (12), pp.3541-3552. ⟨10.1158/0008-5472.CAN-15-1864⟩
LIM kinases (LIMK) are emerging targets for cancer therapy, and they function as network hubs to coordinate actin and microtubule dynamics. When LIMKs are inhibited, actin microfilaments are disorganized and microtubules are stabilized. Owing to thei