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of 5
pro vyhledávání: '"Erika Kajdácsi"'
Autor:
Márta L. Debreczeni, Zsuzsanna Németh, Erika Kajdácsi, Endre Schwaner, Veronika Makó, András Masszi, Zoltán Doleschall, János Rigó, Fruzsina R. Walter, Mária A. Deli, Gábor Pál, József Dobó, Péter Gál, László Cervenak
Publikováno v:
Frontiers in Immunology, Vol 10 (2019)
Pathologically increased vascular permeability is an important dysfunction in the pathomechanism of life-threatening conditions, such as sepsis, ischemia/reperfusion, or hereditary angioedema (HAE), diseases accompanied by uncontrolled activation of
Externí odkaz:
https://doaj.org/article/e45f6f30994a473b8e8f8750cf0088af
Publikováno v:
Clinical Reviews in Allergy & Immunology
In the last few decades, a substantial body of evidence underlined the pivotal role of bradykinin in certain types of angioedema. The formation and breakdown of bradykinin has been studied thoroughly; however, numerous questions remained open regardi
Autor:
Péter Gál, János Rigó, András Masszi, Fruzsina R. Walter, Erika Kajdácsi, László Cervenak, Mária A. Deli, Veronika Makó, Márta L. Debreczeni, Gábor Pál, Zoltán Doleschall, József Dobó, Zsuzsanna Németh, Endre Schwaner
Publikováno v:
Frontiers in Immunology
Frontiers in Immunology, Vol 10 (2019)
Frontiers in Immunology, Vol 10 (2019)
Pathologically increased vascular permeability is an important dysfunction in the pathomechanism of life-threatening conditions, such as sepsis, ischemia/reperfusion, or hereditary angioedema (HAE), diseases accompanied by uncontrolled activation of
Publikováno v:
Clinical Reviews in Allergy & Immunology. 51:140-151
Angioedema, as a distinct disease entity, often becomes a clinical challenge for physicians, because it may cause a life-threatening condition, whereas prompt and accurate laboratory diagnostics may not be available. Although the bedside diagnosis ne
Autor:
László Cervenak, Erika Kajdácsi, Henriette Farkas, Péter K. Jani, Lilian Varga, Zoltán Prohászka, Dorottya Csuka
Publikováno v:
Journal of Allergy and Clinical Immunology. 133:1686-1691
Background Hereditary angioedema (HAE) caused by C1-inhibitor (C1-INH) deficiency (HAE–C1-INH) is a potentially life-threatening rare disease caused by the decreased activity of C1-INH. Lack of C1-INH leads to overproduction of bradykinin, a potent