Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Jiraphun Jittikoon"'
Autor:
Noppadol Chanhom, Jiraphun Jittikoon, Sukanya Wattanapokayakit, Surakameth Mahasirimongkol, Angkana Charoenyingwattana, Wanvisa Udomsinprasert, Usa Chaikledkaew, Supharat Suvichapanich, Taisei Mushiroda, Sasisopin Kiertiburanakul, Archawin Rojanawiwat, Wittaya Wangsomboonsiri, Weerawat Manosuthi, Pacharee Kantipong, Anucha Apisarnthanarak, Wilawan Sangsirinakakul, Pawinee Wongprasit, Romanee Chaiwarith, Woraphot Tantisiriwat, Somnuek Sungkanuparph, Wasun Chantratita
Publikováno v:
Journal of Personalized Medicine, Vol 12, Iss 6, p 940 (2022)
Glutathione s-transferase (GST) is a family of drug-metabolizing enzymes responsible for metabolizing and detoxifying drugs and xenobiotic substances. Therefore, deletion polymorphisms of GSTs can be implicated in developing several pathological cond
Externí odkaz:
https://doaj.org/article/c947fc5618e14a819ae4957935f74913
Autor:
Noppadol Chanhom, Sukanya Wattanapokayakit, Nusara Satproedprai, Supharat Suvichapanich, Surakameth Mahasirimongkol, Usa Chaikledkaew, Wanvisa Udomsinprasert, Taisei Mushiroda, Jiraphun Jittikoon
Publikováno v:
Heliyon, Vol 7, Iss 4, Pp e06852- (2021)
Antituberculosis drug-induced liver injury (ATDILI) is the common adverse reaction of antituberculosis drugs. Glutathione S-transferases (GSTs), which are phase II metabolizing enzymes for detoxification, are recognized as potential mediators of hepa
Externí odkaz:
https://doaj.org/article/8f4f4884bd864234a2ec5801481db618
Autor:
Nusara Satproedprai, Noppadol Chanhom, Usa Chaikledkaew, Jiraphun Jittikoon, Surakameth Mahasirimongkol, Sukanya Wattanapokayakit, Taisei Mushiroda, Wanvisa Udomsinprasert, Supharat Suvichapanich
Publikováno v:
Heliyon
Heliyon, Vol 7, Iss 4, Pp e06852-(2021)
Heliyon, Vol 7, Iss 4, Pp e06852-(2021)
Antituberculosis drug-induced liver injury (ATDILI) is the common adverse reaction of antituberculosis drugs. Glutathione S-transferases (GSTs), which are phase II metabolizing enzymes for detoxification, are recognized as potential mediators of hepa