Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Robert Zeiser"'
Autor:
Asim Saha, Patricia A. Taylor, Christopher J. Lees, Angela Panoskaltsis-Mortari, Mark J. Osborn, Colby J. Feser, Govindarajan Thangavelu, Wolfgang Melchinger, Yosef Refaeli, Geoffrey R. Hill, David H. Munn, Jonathan S. Serody, Ivan Maillard, Katharina Kreymborg, Marcel van den Brink, Chen Dong, Shuyu Huang, Xingxing Zang, James P Allison, Robert Zeiser, Bruce R. Blazar
Publikováno v:
The Journal of Immunology. 202:69.10-69.10
The B7 family members are critical in positive and negative regulation of immune responses by engaging various lymphocyte receptors. B7-H4 is a member of the B7 family that can negatively regulate T cell function. We investigated the role of host and
Autor:
Motoko Koyama, Pamela Mukhopadhyay, Iona S Schuster, Andrea S Henden, Jan Hülsdünker, Antiopi Varelias, Marie Vetizou, Rachel D Kuns, Renee J Robb, Ping Zhang, Bruce R. Blazar, Ranjeny Thomas, Jakob Begun, Nicola Waddell, Giorgio Trinchieri, Robert Zeiser, Andrew D Clouston, Mariapia A Degli-Esposti, Geoffrey R. Hill
Publikováno v:
The Journal of Immunology. 202:69.38-69.38
The factors initiating pathology in the gastrointestinal (GI) tract are largely unknown. Graft-versus-host disease (GVHD) of the gastrointestinal (GI) tract is the principal determinant of lethality following allogeneic bone marrow transplantation (B
Autor:
Christoph Dürr, Michael Sixt, Anna Maria Wolf, Alexander R. Rosenkranz, Lydia Markut, Robert Zeiser, Kathrin Eller, Guenther Gastl, Dominik Wolf, Ulrike V. Gerlach
Publikováno v:
The Journal of Immunology. 183:3751-3760
CD4+CD25+ regulatory T cell (Treg) entry into secondary lymphoid organs and local expansion is critical for their immunosuppressive function. Long-term application of the sphingosine-1 phosphate receptor agonist FTY720 exerts pleiotropic anti-inflamm
Autor:
Dominik von Elverfeldt, Robert S. Negrin, Christoph Dürr, Wilfried Reichardt, Robert Zeiser, Benjie Smith, Ulrike V. Gerlach, Mayumi Yamada, Eva Jüttner
Publikováno v:
The Journal of Immunology. 181:4770-4779
Dendritic cells (DC) play a major role in the pathogenesis of graft-vs-host disease (GvHD). Directed modification of surface molecules on DC that provide instructive signals for T cells may create a tolerogenic DC phenotype that affects GvHD severity
Autor:
Dawn K Reichenbach, Benjamin M Matta, Xiaoli Zhang, Brent Koehn, Lisa Mathews, Michelle J Smith, Colby J Feser, Robert Zeiser, Heth Roderick Turnquist, Bruce R Blazar
Publikováno v:
The Journal of Immunology. 196:140.33-140.33
Graft-versus-host disease (GVHD) is the leading complication and cause of mortality after allogeneic hematopoietic cell transplantation (allo-HCT). Recently, we reported that IL-33 released from tissue damaged during allo-HCT conditioning mediates pr
Autor:
Benjamin Matta, Dawn Reichenbach, Vincent Schwarze, Victor Tkachev, Lisa Mathews, Quan Liu, Max Warncke, James Ferrara, Robert Zeiser, Bruce Blazar, Heth Turnquist
Publikováno v:
The Journal of Immunology. 194:140.13-140.13
Graft-versus-host disease (GVHD) is a severe and often fatal complication of allogeneic (allo) hematopoietic cell transplantation (HCT). Total body irradiation (TBI) conditioning of the recipient and subsequent GVHD-associated alloimmune responses ca
Autor:
Holger Karsunky, Edgar G. Engleman, Irina Fernandez, Kalle Söderström, Andreas Beilhack, Robert S. Negrin, Neeraja Kambham, Robert Zeiser
Publikováno v:
The Journal of Immunology. 179:5605-5605
CD4+CD25+FoxP3+ regulatory T cells (Treg) have been shown to be protective in animal models of autoimmunity and acute graft-vs-host disease. However, owing to the functional heterogeneity among CD4+CD25+ T cells, surface markers expressed selectively
Autor:
Irina Fernandez, Robert Zeiser, Holger Karsunky, Neeraja Kambham, Andreas Beilhack, Kalle Sodestrom, Robert Negrin, Edgar Engleman
Publikováno v:
The Journal of Immunology. 178:S142-S142
CD4+CD25+FoxP3+ regulatory T cells (Treg) have been shown to be protective in animal models of autoimmunity and acute graft-versus-host disease (GVHD). However, owing to the functional heterogeneity among CD4+CD25+ T cells, surface markers expressed