Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Margarete Mehrabian"'
Autor:
Jill K Terra, Bryan France, Christopher K Cote, Amy Jenkins, Joel A Bozue, Susan L Welkos, Ragini Bhargava, Chi-Lee Ho, Margarete Mehrabian, Calvin Pan, Aldons J Lusis, Richard C Davis, Steven M LeVine, Kenneth A Bradley
Publikováno v:
PLoS Pathogens, Vol 7, Iss 12, p e1002469 (2011)
Anthrax is a potentially fatal disease resulting from infection with Bacillus anthracis. The outcome of infection is influenced by pathogen-encoded virulence factors such as lethal toxin (LT), as well as by genetic variation within the host. To ident
Externí odkaz:
https://doaj.org/article/268629d88df4443393b3b819c4cfb5ee
Autor:
Hunter B Fraser, Tomas Babak, John Tsang, Yiqi Zhou, Bin Zhang, Margarete Mehrabian, Eric E Schadt
Publikováno v:
PLoS Genetics, Vol 7, Iss 3, p e1002023 (2011)
The idea that most morphological adaptations can be attributed to changes in the cis-regulation of gene expression levels has been gaining increasing acceptance, despite the fact that only a handful of such cases have so far been demonstrated. Moreov
Externí odkaz:
https://doaj.org/article/693a848fb2e247e29641199010e59916
Autor:
Eric E Schadt, Cliona Molony, Eugene Chudin, Ke Hao, Xia Yang, Pek Y Lum, Andrew Kasarskis, Bin Zhang, Susanna Wang, Christine Suver, Jun Zhu, Joshua Millstein, Solveig Sieberts, John Lamb, Debraj GuhaThakurta, Jonathan Derry, John D Storey, Iliana Avila-Campillo, Mark J Kruger, Jason M Johnson, Carol A Rohl, Atila van Nas, Margarete Mehrabian, Thomas A Drake, Aldons J Lusis, Ryan C Smith, F Peter Guengerich, Stephen C Strom, Erin Schuetz, Thomas H Rushmore, Roger Ulrich
Publikováno v:
PLoS Biology, Vol 6, Iss 5, p e107 (2008)
Genetic variants that are associated with common human diseases do not lead directly to disease, but instead act on intermediate, molecular phenotypes that in turn induce changes in higher-order disease traits. Therefore, identifying the molecular ph
Externí odkaz:
https://doaj.org/article/690823c1095b471bb00d96bfa2f8780e
Autor:
John Lamb, Xia Yang, Erin G. Schuetz, Debraj GuhaThakurta, Aldons J. Lusis, Cliona Molony, Iliana Avila-Campillo, Ke-Ke Hao, Eugene Chudin, Thomas A. Drake, Pek Yee Lum, Jonathan M. J. Derry, Eric E. Schadt, Ryan Smith, Roger G. Ulrich, F. Peter Guengerich, Thomas H. Rushmore, John D. Storey, Jun Zhu, Joshua Millstein, Andrew Kasarskis, Atila van Nas, Bin Zhang, Mark J Kruger, Margarete Mehrabian, Solveig K. Sieberts, Susanna Wang, Jason M. Johnson, Stephen C. Strom, Christine Suver, Carol A. Rohl
Publikováno v:
PLoS Biology, Vol 6, Iss 5, p e107 (2008)
PLoS Biology
PLoS Biology
Genetic variants that are associated with common human diseases do not lead directly to disease, but instead act on intermediate, molecular phenotypes that in turn induce changes in higher-order disease traits. Therefore, identifying the molecular ph