Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Philip J. Norris"'
Autor:
Hilario J. Ramos, Michael Gale, Mehul S. Suthar, Marion C. Lanteri, Piper M. Treuting, Amina Negash, Gabriele Blahnik, Philip J. Norris, Michael P. Busch, Margaret M. Brassil, Khushbu Sodhi
Publikováno v:
PLoS Pathogens, Vol 8, Iss 11, p e1003039 (2012)
PLoS Pathogens
PLoS Pathogens
West Nile virus (WNV) is an emerging flavivirus capable of infecting the central nervous system (CNS) and mediating neuronal cell death and tissue destruction. The processes that promote inflammation and encephalitis within the CNS are important for
Autor:
Alan L. Landay, Cicely Brooks, Elizabeth Sinclair, Philip J. Norris, Joseph M. McCune, Jeffrey N. Martin, Hiroyu Hatano, Michael P. Busch, Peter W. Hunt, Jeffrey Martinson, Brinda Emu, Steven G. Deeks
Publikováno v:
PLoS ONE, Vol 6, Iss 1, p e15924 (2011)
PLoS ONE
PloS one, vol 6, iss 1
PLoS ONE
PloS one, vol 6, iss 1
HIV-infected individuals maintaining undetectable viremia in the absence of therapy (HIV controllers) often maintain high HIV-specific T cell responses, which has spurred the development of vaccines eliciting HIV-specific T cell responses. However, c
Autor:
Leslie H. Tobler, Trevor Peterson, Jonathan L. Bramson, Frank Plummer, Ma Luo, Jacqueline P. Law, Mark Loeb, Zhanna Kaidarova, Michael P. Busch, Shiquan Wu, Marion C. Lanteri, Philip J. Norris, Thomas Bielawny, Steven Cate, Maria Agapova, Brian Custer
Publikováno v:
PLoS ONE, Vol 6, Iss 8, p e22948 (2011)
PLoS ONE
PLoS ONE
Background West Nile virus (WNV) infection is asymptomatic in most individuals, with a minority developing symptoms ranging from WNV fever to serious neuroinvasive disease. This study investigated the impact of host HLA on the outcome of WNV disease.
Autor:
Sheila M. Keating, Emilie Jalbert, Cecilia M. Shikuma, Nisha I. Parikh, Beau K. Nakamoto, Michelle L D'Antoni, Timothy Q. Crawford, Jason D. Barbour, Todd B. Seto, Philip J. Norris, Lishomwa C. Ndhlovu
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e75500 (2013)
PLoS ONE
PLoS ONE
Chronic infection by HIV increases the risk of cardiovascular disease (CVD) despite effective antiretroviral therapy (ART). The mechanisms linking HIV to CVD have yet to be fully elucidated. High plasma levels of the pro-inflammatory cytokine IL-6, w