Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Ulf Hedin"'
Autor:
Sanjay Sinha, Felipe Serrano, Marie A Guerraty, Thomas Quertermous, Yi-An Ko, Milos Pjanic, Wei Zhao, Scott Norton, Yoseph Barash, Daniel J. Rader, Ulf Hedin, John Pluta, Robert C. Wirka, Christopher D. Brown, Trieu Nguyen, Sylvia T. Nurnberg, Ljubica Perisic, Stephanie Testa, Ting Wang, Susannah Elwyn, H. Shanker Rao, YoSon Park
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 16, Iss 1, p e1008538 (2020)
PLoS Genetics, Vol 16, Iss 1, p e1008538 (2020)
Genome-wide association studies have identified multiple novel genomic loci associated with vascular diseases. Many of these loci are common non-coding variants that affect the expression of disease-relevant genes within coronary vascular cells. To i
Functional regulatory mechanism of smooth muscle cell-restricted LMOD1 coronary artery disease locus
Autor:
Ting Wang, Michael Snyder, Thomas Quertermous, Trieu Nguyen, Stephen B. Montgomery, Ulf Hedin, Oscar Franzén, Arno Ruusalepp, Vivek Nanda, Eric E. Schadt, Johan Björkegren, Milos Pjanic, Lijiang Ma, Boxiang Liu, Simon Koplev, Clint L. Miller, Nicholas J. Leeper, Ljubica Perisic Matic
Publikováno v:
PLoS Genetics
PLoS Genetics, Vol 14, Iss 11, p e1007755 (2018)
PLoS Genetics, Vol 14, Iss 11, p e1007755 (2018)
Recent genome-wide association studies (GWAS) have identified multiple new loci which appear to alter coronary artery disease (CAD) risk via arterial wall-specific mechanisms. One of the annotated genes encodes LMOD1 (Leiomodin 1), a member of the ac
Autor:
Jaakko Patrakka, Annika Wernerson, Kjell Hultenby, Mathias Uhlén, Patricia Q. Rodriguez, Timo Pikkarainen, Ying Sun, Christer Betsholtz, Mark Lal, Ljubica Perisic, Karl Tryggvason, Ulf Hedin
Publikováno v:
PLoS ONE
PLoS ONE, Vol 10, Iss 5, p e0126079 (2015)
PLoS ONE, Vol 10, Iss 3, p e0122067 (2015)
PLoS ONE, Vol 10, Iss 5, p e0126079 (2015)
PLoS ONE, Vol 10, Iss 3, p e0122067 (2015)
Background Podocyte foot process effacement accompanied by actin cytoskeleton rearrangements is a cardinal feature of many progressive human proteinuric diseases. Results By microarray profiling of mouse glomerulus, SCHIP1 emerged as one of the most