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Publikováno v:
Carcinogenesis, 39(11), 1342-1351. Oxford University Press
Hahn, Markus; Simons, Colinda C J M; Weijenberg, Matty P; van den Brandt, Piet A (2018). Alcohol drinking, ADH1B and ADH1C genotypes and the risk of postmenopausal breast cancer by hormone receptor status: the Netherlands Cohort Study on diet and cancer. Carcinogenesis, 39(11), pp. 1342-1351. Oxford University Press 10.1093/carcin/bgy101
Hahn, Markus; Simons, Colinda C J M; Weijenberg, Matty P; van den Brandt, Piet A (2018). Alcohol drinking, ADH1B and ADH1C genotypes and the risk of postmenopausal breast cancer by hormone receptor status: the Netherlands Cohort Study on diet and cancer. Carcinogenesis, 39(11), pp. 1342-1351. Oxford University Press 10.1093/carcin/bgy101
Alcohol consumption has consistently been shown to increase breast cancer (BC) risk. This association may be modified by single nucleotide polymorphisms (SNPs) in alcohol dehydrogenase (ADH) isoenzymes ADH1B and ADH1C. The Netherlands Cohort Study co
Autor:
Patricia A. Ganz
Publikováno v:
J Natl Cancer Inst
JNCI Journal of the National Cancer Institute
JNCI Journal of the National Cancer Institute
Background The American Cancer Society, Centers for Disease Control and Prevention, National Cancer Institute, and North American Association of Central Cancer Registries provide annual updates on cancer occurrence and trends by cancer type, sex, rac
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::95cb857352f602040d9a6ae2ce7e5e9b
https://europepmc.org/articles/PMC6910157/
https://europepmc.org/articles/PMC6910157/
Autor:
Meeks, H.D., Song, H.L., Michailidou, K., Bolla, M.K., Dennis, J., Wang, Q., Barrowdale, D., Frost, D., McGuffog, L., Ellis, S., Feng, B.J., Buys, S.S., Hopper, J.L., Southey, M.C., Tesoriero, A., James, P.A., Bruinsma, F., Campbell, I.G., Broeks, A., Schmidt, M.K., Hogervorst, F.B.L., Beckman, M.W., Fasching, P.A., Fletcher, O., Johnson, N., Sawyer, E.J., Riboli, E., Banerjee, S., Menon, U., Tomlinson, I., Burwinkel, B., Hamann, U., Marme, F., Rudolph, A., Janavicius, R., Tihomirova, L., Tung, N., Garber, J., Cramer, D., Terry, K.L., Poole, E.M., Tworoger, S.S., Dorfling, C.M., Rensburg, E.J. van, Godwin, A.K., Guenel, P., Truong, T., Stoppa-Lyonnet, D., Damiola, F., Mazoyer, S., Sinilnikova, O.M., Isaacs, C., Maugard, C., Bojesen, S.E., Flyger, H., Gerdes, A.M., Hansen, T.V.O., Jensen, A., Kjaer, S.K., Hogdall, C., Hogdall, E., Pedersen, I.S., Thomassen, M., Benitez, J., Gonzalez-Neira, A., Osorio, A., Hoya, M. de la, Segura, P.P., Diez, O., Lazaro, C., Brunet, J., Anton-Culver, H., Eunjung, L., John, E.M., Neuhausen, S.L., Ding, Y.C., Castillo, D., Weitzel, J.N., Ganz, P.A., Nussbaum, R.L., Chan, S.B., Karlan, B.Y., Lester, J., Wu, A., Gayther, S., Ramus, S.J., Sieh, W., Whittermore, A.S., Monteiro, A.N.A., Phelan, C.M., Terry, M.B., Piedmonte, M., Offit, K., Robson, M., Levine, D., Moysich, K.B., Cannioto, R., Olson, S.H., Daly, M.B., Nathanson, K.L., Domchek, S.M., Lu, K.H., Liang, D., Hildebrant, M.A.T., Ness, R., Modugno, F., Pearce, L., Goodman, M.T., Thompson, P.J., Brenner, H., Butterbach, K., Meindl, A., Hahnen, E., Wappenschmidt, B., Brauch, H., Bruning, T., Blomqvist, C., Khan, S., Nevanlinna, H., Pelttari, L.M., Aittomaki, K., Butzow, R., Bogdanova, N.V., Dork, T., Lindblom, A., Margolin, S., Rantala, J., Kosma, V.M., Mannermaa, A., Lambrechts, D., Neven, P., Claes, K.B.M., Maerken, T. van, Chang-Claude, J., Flesch-Janys, D., Heitz, F., Varon-Mateeva, R., Peterlongo, P., Radice, P., Viel, A., Barile, M., Peissel, B., Manoukian, S., Montagna, M., Oliani, C., Peixoto, A., Teixeira, M.R., Collavoli, A., Hallberg, E., Olson, J.E., Goode, E.L., Hart, S.N., Shimelis, H., Cunningham, J.M., Giles, G.G., Milne, R.L., Healey, S., Tucker, K., Haiman, C.A., Henderson, B.E., Goldberg, M.S., Tischkowitz, M., Simard, J., Soucy, P., Eccles, D.M., N. le, Borresen-Dale, A.L., Kristensen, V., Salvesen, H.B., Bjorge, L., Bandera, E.V., Risch, H., Zheng, W., Beeghly-Fadiel, A., Cai, H., Pylkas, K., Tollenaar, R.A.E.M., Ouweland, A.M.W. van der, Andrulis, I.L., Knight, J.A., Narod, S., Devilee, P., Winqvist, R., Figueroa, J., Greene, M.H., Mai, P.L., Loud, J.T., Garcia-Closas, M., Schoemaker, M.J., Czene, K., Darabi, H., McNeish, I., Siddiquil, N., Glasspool, R., Kwong, A., Park, S.K., Teo, S.H., Yoon, S.Y., Matsuo, K., Hosono, S., Woo, Y.L., Gao, Y.T., Foretova, L., Singer, C.F., Rappaport-Feurhauser, C., Friedman, E., Laitman, Y., Rennert, G., Imyanitov, E.N., Hulick, P.J., Olopade, O.I., Senter, L., Olah, E., Doherty, J.A., Schildkraut, J., Koppert, L.B., Kiemeney, L.A., Massuger, L.F.A.G., Cook, L.S., Pejovic, T., Li, J.M., Borg, A., Ofverholm, A., Rossing, M.A., Wentzensen, N., Henriksson, K., Cox, A., Cross, S.S., Pasini, B.J., Shah, M., Kabisch, M., Torres, D., Jakubowska, A., Lubinski, J., Gronwald, J., Agnarsson, B.A., Kupryjanczyk, J., Moes-Sosnowska, J., Fostira, F., Konstantopoulou, I., Slager, S., Jones, M., Antoniou, A.C., Berchuck, A., Swerdlow, A., Chenevix-Trench, G., Dunning, A.M., Pharoah, P.D.P., Hall, P., Easton, D.F., Couch, F.J., Spurdle, A.B., Goldgar, D.E., EMBRACE, kConFab Investigators, Australia Ovarian Canc Study Grp, HEBON, GEMO Study Collaborators, OCGN, PRostate Canc Assoc Grp
Publikováno v:
Meeks, H D, Song, H, Michailidou, K, Bolla, M K, Dennis, J, Wang, Q, Barrowdale, D, Frost, D, EMBRACE, McGuffog, L, Ellis, S, Feng, B, Buys, S S, Hopper, J L, Southey, M C, Tesoriero, A, kConFab Investigators, James, P A, Bruinsma, F, Campbell, I G, Broeks, A, Schmidt, M K, Hogervorst, F B L, HEBON, Beckman, M W, Fasching, P A, Fletcher, O, Johnson, N, Sawyer, E J, Riboli, E, Banerjee, S, Menon, U, Tomlinson, I, Burwinkel, B, Hamann, U, Marme, F, Rudolph, A, Janavicius, R, Tihomirova, L, Tung, N, Garber, J, Cramer, D, Terry, K L, Poole, E M, Tworoger, S S, Dorfling, C M, van Rensburg, E J, Godwin, A K, Guénel, P, Truong, T, GEMO Study Collaborators, Stoppa-Lyonnet, D, Damiola, F, Pedersen, I S, OCGN & PRostate cancer AssoCiation group To Investigate Cancer Associated aLterations in the genome 2016, ' BRCA2 Polymorphic Stop Codon K3326X and the Risk of Breast, Prostate, and Ovarian Cancers ', National Cancer Institute. Journal, vol. 108, no. 2, djv315 . https://doi.org/10.1093/jnci/djv315
Journal of the National Cancer Institute, 108
Journal of the National Cancer Institute, 108(2):315. Oxford University Press
Journal of the National Cancer Institute, 108(2):djv315. Oxford University Press
Journal of the National Cancer Institute, 108, 2
JNCI: Journal of the National Cancer Institute, 108(2)
Meeks, H D, Song, H, Michailidou, K, Bolla, M K, Dennis, J, Wang, Q, Barrowdale, D, Frost, D, McGuffog, L, Ellis, S, Feng, B, Buys, S S, Hopper, J L, Southey, M C, Tesoriero, A, James, P A, Bruinsma, F, Campbell, I G, Broeks, A, Schmidt, M K, Hogervorst, F B L, Beckman, M W, Fasching, P A, Fletcher, O, Johnson, N, Sawyer, E J, Riboli, E, Banerjee, S, Menon, U, Tomlinson, I, Burwinkel, B, Hamann, U, Marme, F, Rudolph, A, Janavicius, R, Tihomirova, L, Tung, N, Garber, J, Cramer, D, Terry, K L, Poole, E M, Tworoger, S S, Dorfling, C M, van Rensburg, E J, Godwin, A K, Guénel, P, Truong, T, Stoppa-Lyonnet, D, Gerdes, A-M, Thomassen, M & EMBRACE 2016, ' BRCA2 Polymorphic Stop Codon K3326X and the Risk of Breast, Prostate, and Ovarian Cancers ', Journal of the National Cancer Institute, vol. 108, no. 2, djv315 . https://doi.org/10.1093/jnci/djv315
Journal of the National Cancer Institute, 108
Journal of the National Cancer Institute, 108(2):315. Oxford University Press
Journal of the National Cancer Institute, 108(2):djv315. Oxford University Press
Journal of the National Cancer Institute, 108, 2
JNCI: Journal of the National Cancer Institute, 108(2)
Meeks, H D, Song, H, Michailidou, K, Bolla, M K, Dennis, J, Wang, Q, Barrowdale, D, Frost, D, McGuffog, L, Ellis, S, Feng, B, Buys, S S, Hopper, J L, Southey, M C, Tesoriero, A, James, P A, Bruinsma, F, Campbell, I G, Broeks, A, Schmidt, M K, Hogervorst, F B L, Beckman, M W, Fasching, P A, Fletcher, O, Johnson, N, Sawyer, E J, Riboli, E, Banerjee, S, Menon, U, Tomlinson, I, Burwinkel, B, Hamann, U, Marme, F, Rudolph, A, Janavicius, R, Tihomirova, L, Tung, N, Garber, J, Cramer, D, Terry, K L, Poole, E M, Tworoger, S S, Dorfling, C M, van Rensburg, E J, Godwin, A K, Guénel, P, Truong, T, Stoppa-Lyonnet, D, Gerdes, A-M, Thomassen, M & EMBRACE 2016, ' BRCA2 Polymorphic Stop Codon K3326X and the Risk of Breast, Prostate, and Ovarian Cancers ', Journal of the National Cancer Institute, vol. 108, no. 2, djv315 . https://doi.org/10.1093/jnci/djv315
Contains fulltext : 172007.pdf (Publisher’s version ) (Closed access) BACKGROUND: The K3326X variant in BRCA2 (BRCA2*c.9976A>T; p.Lys3326*; rs11571833) has been found to be associated with small increased risks of breast cancer. However, it is not
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cb23f08642717d4e65f3010a25475b41
https://eprints.gla.ac.uk/117662/1/117662.pdf
https://eprints.gla.ac.uk/117662/1/117662.pdf
BACKGROUND The Affordable Care Act (ACA) removed copayments for screening mammography and colonoscopy in Medicare beneficiaries, but its clinical impact is unknown. METHODS Using a 5% random sample of Medicare claims from 2009 through 2012 in individ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::dc08f65db8cccb89203105d7b5a1eb7b
https://europepmc.org/articles/PMC4849805/
https://europepmc.org/articles/PMC4849805/
Background Years of potential life lost (YPLL) complement incidence and survival rates by measuring how much a patient's life is likely to be shortened by his or her cancer. In this study, we examine the impact of death due to brain and other central
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a235857d9a8d4a539aaf931aadd18dbd
https://europepmc.org/articles/PMC4677421/
https://europepmc.org/articles/PMC4677421/
Autor:
Claire Taylor, David G. Cox, Faye Elliott, Anne E. Kiltie, Stefano Landi, Janet Hall, Debora Landi, Mark Teo, Laurence Vaslin, D. Timothy Bishop
Publikováno v:
Carcinogenesis
MicroRNAs (miRNAs) are involved in post-transcriptional regulation of gene expression through binding to messenger RNAs (mRNA) thereby promoting mRNA degradation or altered translation. A single-nucleotide polymorphism (SNP) located within a miRNA-bi
Autor:
Recinda L. Sherman, Ahmedin Jemal, Lynne Penberthy, Robert N. Anderson, A. Blythe Ryerson, Francis P. Boscoe, Anne-Michelle Noone, S. Jane Henley, Nadia Howlader, Andrew C Lake, Betsy A. Kohler, Kevin A. Henry, Kathleen A. Cronin, Christie R. Eheman
Publikováno v:
JNCI Journal of the National Cancer Institute
Background: The American Cancer Society (ACS), Centers for Disease Control and Prevention (CDC), National Cancer Institute (NCI), and North American Association of Central Cancer Registries (NAACCR) collaborate annually to produce updated, national c
Autor:
Kim Anh Do, Argun Akcakanat, Stacy Moulder-Thompson, Razelle Kurzrock, Ian E. Krop, Laurence A. Doyle, Gordon B. Mills, Yisheng Li, Kirk S. Culotta, Emily Tarco, Aysegul A. Sahin, Funda Meric-Bernstam, Eric P. Winer, Ana M. Gonzalez-Angulo, Sarina Anne Piha-Paul, Vivianne Velez-Bravo, Huiqin Chen, Shuying Liu
Publikováno v:
Gonzalez-Angulo, AM; Krop, I; Akcakanat, A; Chen, H; Liu, S; Li, Y; et al.(2015). SU2C phase Ib study of paclitaxel and MK-2206 in advanced solid tumors and metastatic breast cancer.. Journal of the National Cancer Institute, 107(3). doi: 10.1093/jnci/dju493. UC San Diego: Retrieved from: http://www.escholarship.org/uc/item/8cw660c5
Journal of the National Cancer Institute, vol 107, iss 3
Journal of the National Cancer Institute, vol 107, iss 3
BACKGROUND:There is preclinical synergism between taxanes and MK-2206. We aim to determine the maximum tolerated dose, safety, and activity of combining MK-2206 and paclitaxel in metastatic cancer. METHODS:Patients received weekly doses of paclitaxel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e90bd7024a95bec19908348956811bb2
https://europepmc.org/articles/PMC4342675/
https://europepmc.org/articles/PMC4342675/
Autor:
Mark Clendenning, John L. Hopper, Barbara A. Leggett, Daniel D. Buchanan, Mark A. Jenkins, Steven Gallinger, Noralane M. Lindor, A. Joan Levine, Graeme P. Young, Elizabeth A. Williamson, Jack Goldblatt, Robert W. Haile, Lara Lipton, Ingrid Winship, Joanne P. Young, Polly A. Newcomb, Katherine M. Tucker, Aung Ko Win, Christophe Rosty, Loïc Le Marchand, Graham G. Giles, Finlay A. Macrae, Susan Parry, Julie Arnold
Background L ynch syndrome is a highly penetrant cancer predisposition syndrome caused by germline mutations in DNA mismatch repair (MMR) genes. We estimated the risks of primary cancers other than colorectal cancer following a diagnosis of colorecta
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b5066842686e0d4f8cd0803310f231e
https://europepmc.org/articles/PMC3529597/
https://europepmc.org/articles/PMC3529597/
Autor:
Norman Daniels, Sue J. Goldie
Disease simulation models of the health and economic consequences of different prevention and treatment strategies can guide policy decisions about cancer control. However, models that also consider health disparities can identify strategies that imp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50cfb7ccc3105ef30b84ff9e1b518e5e
https://europepmc.org/articles/PMC3176779/
https://europepmc.org/articles/PMC3176779/