Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Motohiro Nishida"'
Autor:
Hitoshi Kurose, Takahiro Iwamoto, Mayumi Hirano, Ryuji Inoue, Michio Nakaya, Zhong Jian, Shota Saiki, Motohiro Nishida, Yoji Sato, Satomi Kita, Marina Ariyoshi, Kinue Nishioka, Katsuya Hirano
Publikováno v:
Arteriosclerosis, Thrombosis, and Vascular Biology. 31:2278-2286
Objective— The goal of this study was to determine whether inhibition of transient receptor potential canonical (TRPC) channels underlies attenuation of angiotensin II (Ang II)–induced vasoconstriction by phosphodiesterase (PDE) 3 inhibition. Met
Autor:
Masataka Fujiwara, Yoshihiro Kuwabara, Zhong Jian, Hideyuki Kinoshita, Xianglu Rong, Yuhao Li, Kazuwa Nakao, Koichiro Kuwahara, Yasuo Mori, Motohiro Nishida, Yuko Yamada, Satoru Usami, Ryuji Inoue, Kenji Ueshima, Hitoshi Kurose, Yasuaki Nakagawa, Takeya Minami, Shigeki Kiyonaka
Publikováno v:
Circulation Research. 106:1849-1860
Rationale : Atrial and brain natriuretic peptides (ANP and BNP, respectively) exert antihypertrophic effects in the heart via their common receptor, guanylyl cyclase (GC)-A, which catalyzes the synthesis of cGMP, leading to activation of protein kina
Autor:
Hitoshi Kurose, Motohiro Nishida, Taku Nagao, Tohru Kozasa, Justin H. Turner, Yoshiko Maruyama, Shihori Tanabe, Teiji Wada, Yoshiyuki Sugimoto
Publikováno v:
Circulation Research. 91:961-969
In neonatal cardiomyocytes, activation of the G q -coupled α 1 -adrenergic receptor (α 1 AR) induces hypertrophy by activating mitogen-activated protein kinases, including c-Jun NH 2 -terminal kinase (JNK). Here, we show that JNK activation is esse
Autor:
Hideyuki Kinoshita, R. Inoue, H Kurose, Koichiro Kuwahara, Motohiro Nishida, Kazuwa Nakao, Y Mori
Publikováno v:
Journal of Hypertension. 28:e352