Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Hisashi Oishi"'
Autor:
Fumiko Tomiyama, Takaya Suzuki, Tatsuaki Watanabe, Jun Miyanaga, Anna Suzuki, Takayasu Ito, Sho Murai, Yuyo Suzuki, Hiromichi Niikawa, Hisashi Oishi, Hirotsugu Notsuda, Yui Watanabe, Takashi Hirama, Ken Onodera, Takeo Togo, Masafumi Noda, Thomas K. Waddell, Golnaz Karoubi, Yoshinori Okada
Publikováno v:
Scientific Reports, Vol 14, Iss 1, Pp 1-12 (2024)
Abstract Whole lung engineering and the transplantation of its products is an ambitious goal and ultimately a viable solution for alleviating the donor-shortage crisis for lung transplants. There are several limitations currently impeding progress in
Externí odkaz:
https://doaj.org/article/52c96afc31ac441187e69c4ada4801ca
Autor:
Nahoko Fujimoto, Katsuya Nagaoka, Ichiro Tatsuno, Hisashi Oishi, Makoto Tomita, Tadao Hasegawa, Yasuhito Tanaka, Takahiro Matsumoto
Publikováno v:
Scientific Reports, Vol 13, Iss 1, Pp 1-7 (2023)
Abstract Ultraviolet (UV) irradiation offers an effective and convenient method for the disinfection of pathogenic microorganisms. However, UV irradiation causes protein and/or DNA damage; therefore, further insight into the performance of different
Externí odkaz:
https://doaj.org/article/9a26ff5864944881bd4da5f6dac59583
Autor:
Naoya Yamaguchi, Atsushi Suzuki, Aya Yoshida, Tatsushi Tanaka, Kohei Aoyama, Hisashi Oishi, Yuichiro Hara, Tomoo Ogi, Izuki Amano, Satomi Kameo, Noriyuki Koibuchi, Yasuhiro Shibata, Shinya Ugawa, Haruo Mizuno, Shinji Saitoh
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-9 (2022)
Abstract SLC26A4 is a known iodide transporter, and is localized at the apical membrane of thyrocytes. Previously, we reported that SLC26A7 is also involved in iodide transport and that Slc26a7 is a novel causative gene for congenital hypothyroidism.
Externí odkaz:
https://doaj.org/article/e96f5e5bc5174803af6db215698f5947
Autor:
Mai Thi Nhu Tran, Michito Hamada, Hyojung Jeon, Risako Shiraishi, Keigo Asano, Motochika Hattori, Megumi Nakamura, Yuki Imamura, Yuki Tsunakawa, Risa Fujii, Toshiaki Usui, Kaushalya Kulathunga, Christina-Sylvia Andrea, Ryusuke Koshida, Risa Kamei, Yurina Matsunaga, Makoto Kobayashi, Hisashi Oishi, Takashi Kudo, Satoru Takahashi
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-14 (2017)
Complement component C1q activates efferocytosis, suppresses inflammatory responses, and is thereby thought to limit autoimmune disease. Here, the authors show that macrophage transcription factor MafB regulates total serum levels of C1q, which contr
Externí odkaz:
https://doaj.org/article/8ad6548fe15640be8a3fcc672a1b03f2
Autor:
Risa Kamei, Christina-Sylvia Andrea, Risa Fujii, Makoto Kobayashi, Motochika Hattori, Megumi Nakamura, Keigo Asano, Mai Thi Nhu Tran, Ryusuke Koshida, Yuki Imamura, Michito Hamada, Satoru Takahashi, Hyojung Jeon, Takashi Kudo, Hisashi Oishi, Kaushalya Kulathunga, Yurina Matsunaga, Toshiaki Usui, Yuki Tsunakawa, Risako Shiraishi
Publikováno v:
Nature Communications, Vol 8, Iss 1, Pp 1-14 (2017)
Nature Communications
Nature Communications
The transcription factor MafB is expressed by monocytes and macrophages. Efferocytosis (apoptotic cell uptake) by macrophages is important for inhibiting the development of autoimmune diseases, and is greatly reduced in Mafb-deficient macrophages. He