Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Riccardo Brambilla"'
Publikováno v:
Neuroscience & Biobehavioral Reviews. 142:104892
Here we discuss the role of diverse environmental manipulations affecting cognition with special regard to psychiatric conditions. We present evidence supporting a direct causal correlation between the valence of the environmental stimulation and som
Publikováno v:
Neurobiology of Learning and Memory. 105:63-80
The striatum is the input structure of the basal ganglia system. By integrating glutamatergic signals from cortical and subcortical regions and dopaminergic signals from mesolimbic nuclei the striatum functions as an important neural substrate for pr
Publikováno v:
Journal of Neuroscience Methods. 185:1-14
The use of viral vectors as gene transfer tools for the central nervous system has seen a significant growth in the last decade. Improvements in the safety, efficiency and specificity of vectors for clinical applications have proven to be beneficial
Publikováno v:
Neurobiology of Learning and Memory. 124:1-2
Autor:
Annamaria Porrazzo, Riccardo Brambilla, Vincenzo Cestari, Jacques Pouysségur, Wojciech Krezel, Anne Marowsky, Stefania Fasano, Hans Welzl, Paul C. Orban, Chiara Vantaggiato, Alessandro Ciamei, Paul F. Chapman, Olga Valverde, Rafael Maldonado, Hans-Peter Lipp, Cristina Mazzucchelli, Pavel Pakhotin, Markus U. Ehrengruber, David P. Wolfer, Gilles Pagès
Publikováno v:
Neuron. 34:807-820
Extracellular signal-regulated kinases (ERK1 and 2) are synaptic signaling components necessary for several forms of learning. In mice lacking ERK1, we observe a dramatic enhancement of striatum-dependent long-term memory, which correlates with a fac
Autor:
Rüdiger Klein, Riccardo Brambilla
Publikováno v:
Molecular and Cellular Neuroscience. 6:487-495
Autor:
Ubaldo Del Carro, Claudio Bordignon, Angelo Quattrini, Alessia Capotondo, Hisaya Azuma, Sergio Marchesini, Riccardo Brambilla, Alessandra Biffi, Luigi Naldini, Markus Grompe, Stefania Fasano
Publikováno v:
Molecular Therapy. 13
Metachromatic Leukodystrophy (MLD) is a demyelinating lysosomal storage disorder with an urgent medical need. We previously showed that transplantation of lentiviral vector- transduced Hematopoietic Stem Cells (HSC) targets therapeutic genes to the C
Autor:
Ubaldo Del Carro, Alessia Capotondo, Luigi Naldini, Marcus Grompe, Riccardo Brambilla, Angelo Quattrini, Sergio Marchesini, Claudio Bordignon, Alessandra Biffi, Stefania Fasano
Publikováno v:
Molecular Genetics and Metabolism. 92:14-15
Autor:
Ubaldo Del Carro, Alessandra Biffi, Luigi Naldini, Angelo Quattrini, Ilaria Bertani, Ilaria Visigalli, Michele De Palma, Claudio Bordignon, Riccardo Brambilla, Sergio Marchesini
Publikováno v:
Molecular Therapy. 9:S17-S18
MLD and GLD are inborn lipidosis caused by the deficiency of the lysosomal enzymes arylsulfatase A (ARSA) and galactocerebrosidase (GALC). They are characterized by storage of undegraded sulfatides in the CNS and PNS, leading to progressive demyelina
Autor:
Angelo Quattrini, Alessia Capotondo, Riccardo Brambilla, Stefania Fasano, Claudio Bordignon, Alessandra Biffi, Luigi Naldini, Ubaldo Del Carro, Sergio Marchesini
Publikováno v:
Molecular Therapy. 11:S165
Top of pageAbstract Metachromatic Leukodystrophy (MLD) is an inborn lipidosis caused by deficiency of the lysosomal enzyme arylsulfatase A (ARSA). It is characterized by storage of undegraded sulfatides in the CNS and PNS, leading to severe demyelina