Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Alessandro Stella"'
Autor:
Agostino Di Ciaula, Matteo Iacoviello, Leonilde Bonfrate, Mohamad Khalil, Harshitha Shanmugam, Giuseppe Lopalco, Rosanna Bagnulo, Antonella Garganese, Florenzo Iannone, Nicoletta Resta, Piero Portincasa, Alessandro Stella
Publikováno v:
European Journal of Internal Medicine.
Autor:
Vincenzo Venerito, Alessandro Stella, Gerardo Cazzato, Antonietta Cimmino, Giuseppe Lopalco, Florenzo Iannone, Piero Portincasa
Publikováno v:
Joint Bone Spine. 89:105259
Publikováno v:
Tetrahedron Letters. 54:830-833
A facile and efficient synthesis of 6-amino and 6-aminoalkyl thiazolo[4,5-c]pyridazines is reported. The key step for the construction of this novel bicyclic scaffold was the reaction between 3-amino-4-bromopyridazine derivatives and alkylisothiocyan
Autor:
Francesco Papa, Francesca Paola Lorusso, Anna Maria Sardanelli, Patrizio Panelli, Alessandro Stella, Damiano Panelli, Sergio Papa, Massimo Caputi
Publikováno v:
Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms. 1829:211-218
A study is presented on the regulation of alternative splicing (AS) of the Ndufb11 gene of complex I of the mitochondrial respiratory chain and the impact on this process of rotenone treatment in neuroblastoma cells. In physiological conditions the N
Autor:
Maria Trojano, Paolo Livrea, Alessandro Stella, Carlo Avolio, Ginevra Guanti, F. De Robertis, Maria Liguori
Publikováno v:
Journal of the Neurological Sciences. 162:194-200
We conducted a comparative analysis of clinical and demographic findings between pairs of relatives (36 sibling and 9 parent/child), concordant for Multiple Sclerosis (MS), from 40 MS Italian Multiplex families. A genetic TNF (alpha and beta) loci ty
Autor:
Mattia Gentile, Paola Origoni, Angelo Lonoce, Nicoletta Resta, Maria Pina Montero, Cristina Mareni, Alessandro Stella, Giuseppina Brescia, Francesco Susca, Ginevra Guanti
Publikováno v:
Biochemical and Biophysical Research Communications. 184:1357-1363
Summary Familial Adenomatous Polyposis (FAP) is a premalignant disease of the gastrointestinal tract inherited as an autosomal dominant trait assigned to chromosome 5q21. The 15 exons of the APC gene responsible for the defect were amplified from the