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Autor:
Robert J. Bridges, Fuquan Chen, Yanying Dai, Steven M. Rowe, Xiaojiao Xue, David M. Bedwell, Ming Du, Jeong S. Hong, Valery Belakhov, Jochen Schacht, Timor Baasov, Venkateshwar Mutyam, Liping Tang, Moran Shalev, Silpak Biswas, Laura A. Jackson
New drugs are needed to enhance premature termination codon (PTC) suppression to treat the underlying cause of cystic fibrosis (CF) and other diseases caused by nonsense mutations. We tested new synthetic aminoglycoside derivatives expressly develope
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0fd60151a0e171101e0cc9f6d6da01e6
https://europepmc.org/articles/PMC4068923/
https://europepmc.org/articles/PMC4068923/
Publikováno v:
American Journal of Respiratory and Critical Care Medicine. 153:1116-1122
It has been suggested that surfactant protein-A (SP-A) protects surfactant activity from inhibitors such as fibrinogen. Substantial evidence indicates that inhibition of surfactant activity is often important in the pathogenesis of acute respiratory