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Autor:
Gerrit van Hall, Thomas Idorn, Bo Feldt-Rasmussen, Iain Bressendorff, Bolette Hartmann, Casper Rydahl, Nicolai J. Wewer Albrechtsen, Lisbet Brandi, Jens J. Holst, Henrik Post Hansen, Morten B. Jørgensen, Mads Hornum, Filip K. Knop
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 105:e564-e574
Context The insulin-stimulating and glucagon-regulating effects of the 2 incretin hormones, glucagon-like peptide 1 (GLP-1) and glucose-dependent insulinotropic polypeptide (GIP), contribute to maintain normal glucose homeostasis. Impaired glucose to
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 100:3787-3799
Fifty percent of pediatric low-grade gliomas affect the optic pathway, hypothalamus, and suprasellar areas (OP/HSGs), resulting in significant long-term neuroendocrinopathy.This study aimed to dissect tumor- from treatment-related risk factors for OP
Autor:
Ian F. Godsland, Ali Alsafi, Rachel C. Troke, Victoria Salem, Mandy Donaldson, Mohammad A. Ghatei, Kevin C.R. Baynes, James Minnion, Benjamin C. T. Field, Stephen R. Bloom, Tricia Tan, Akila De Silva, Shivani Misra
Publikováno v:
E2324
E2317
The Journal of Clinical Endocrinology and Metabolism
E2317
The Journal of Clinical Endocrinology and Metabolism
Context: The combination of peptide YY (PYY) and glucagon-like peptide-1 (GLP-1) has been proposed as a potential treatment for diabetes and obesity. However, the combined effects of these hormones, PYY3–36 and GLP-17–36 amide, on glucose homeost
Autor:
Susanna Leskelä, Ana Rodríguez-Muñoz, Hortensia de la Fuente, Francisco Sánchez-Madrid, Pedro Bonay, Mónica Marazuela, Nicté Figueroa-Vega, Roberto González-Amaro, Pilar Martín, Ana Serrano
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 98:2822-2833
Patients with autoimmune thyroid diseases (AITD) show defects in immunoregulatory mechanisms. Herein we assessed the expression of different regulatory receptors in circulating and thyroid dendritic cells (DCs).Peripheral blood samples from 49 patien
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 97:1363-1370
The insulinotropic effect of the incretin hormones, glucose-dependent insulinotropic polypeptide (GIP), and glucagon-like peptide-1 (GLP-1) is impaired in patients with type 2 diabetes. It remains unclear whether this impairment is a primary pathophy
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 96:1283-1291
Specimens have thyroglobulin antibody (TgAb) measured prior to thyroglobulin (Tg) testing because the qualitative TgAb status (positive or negative) determines risk for Tg assay interference, and the quantitative TgAb concentration serves as a surrog
Autor:
Shabina Roohi Ahmed, Douglas W. Ball
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 96:1237-1245
Delay in the diagnosis of medullary thyroid cancer until after thyroidectomy is relatively common and leads to suboptimal treatment.
Autor:
Vincent Col, Maximin Senou, Matthieu Thimmesch, Anne-Catherine Gérard, Marie-Christine Many, Olivier Devuyst, Jean-Nicolas Audinot, François Jouret, Pascale Lipnik, Ides M. Colin, Jacques Emile Dumont, José Moreno, Jacqueline Van Sande, Céline Khalifa
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism; Vol 95
The Journal of Clinical Endocrinology & Metabolism
The Journal of Clinical Endocrinology & Metabolism
Context: Pendred syndrome is caused by mutations in the gene coding for pendrin, an apical Cl(-)/I(-) exchanger. Objective: To analyze intrathyroidal compensatory mechanisms when pendrin is lacking, we investigated the thyroid of a patient with Pendr
Autor:
Victor J. Bernet
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 95:3586-3592
Analysis of the Surveillance Epidemiology and End Results database reveals that since 1995 a 2.4-fold increase in thyroid cancer has occurred. A concomitant rise in cases of thyroid microcarcinoma has also been noted, with the frequency rising by app
Publikováno v:
The Journal of Clinical Endocrinology & Metabolism. 95:2529-2535
Amiodarone, a benzofuranic iodine-rich antiarrhythmic drug, causes thyroid dysfunction in 15-20% of cases. Although amiodarone-induced hypothyroidism poses no particular problem, amiodarone-induced thyrotoxicosis (AIT) is a diagnostic and therapeutic