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Publikováno v:
Retrovirology, Vol 16, Iss 1, Pp 1-14 (2019)
Retrovirology
Retrovirology
Human T-cell leukemia virus type-1 (HTLV-1) causes adult T-cell leukemia/lymphoma (ATL), HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP), and other inflammatory diseases. There is no disease-specific difference in viral strains, a
Autor:
Peter A. Barry, Andrew A. Lackner, Pyone P. Aye, Janice E. Clements, Haitao Hu, Lucio Gama, Drew Weissman
Publikováno v:
Retrovirology, Vol 8, Iss 1, p 57 (2011)
Retrovirology
Hu, Haitao; Gama, Lucio; Aye, Pyone P; Clements, Janice E; Barry, Peter A; Lackner, Andrew A; et al.(2011). SIV antigen immunization induces transient antigen-specific T cell responses and selectively activates viral replication in draining lymph nodes in retroviral suppressed rhesus macaques. Retrovirology, 8(1), 57. doi: http://dx.doi.org/10.1186/1742-4690-8-57. Retrieved from: http://www.escholarship.org/uc/item/3j49q153
Retrovirology
Hu, Haitao; Gama, Lucio; Aye, Pyone P; Clements, Janice E; Barry, Peter A; Lackner, Andrew A; et al.(2011). SIV antigen immunization induces transient antigen-specific T cell responses and selectively activates viral replication in draining lymph nodes in retroviral suppressed rhesus macaques. Retrovirology, 8(1), 57. doi: http://dx.doi.org/10.1186/1742-4690-8-57. Retrieved from: http://www.escholarship.org/uc/item/3j49q153
Background HIV infection causes a qualitative and quantitative loss of CD4+ T cell immunity. The institution of anti-retroviral therapy (ART) restores CD4+ T cell responses to many pathogens, but HIV-specific responses remain deficient. Similarly, th
Autor:
Francesca Tarabella, Leonia Bozzacco, Mauro Bendinelli, Mauro Pistello, Antonio Merico, Luca Ceccherini-Nelli, Paola Mazzetti, Donatella Matteucci, Giulia Freer, Enrica Ricci
Publikováno v:
Retrovirology, Vol 5, Iss 1, p 33 (2008)
Retrovirology
Retrovirology
Immunotherapy of feline immunodeficiency virus (FIV)-infected cats with monocyte-derived dendritic cells (MDCs) loaded with aldrithiol-2 (AT2)-inactivated homologous FIV was performed. Although FIV-specific lymphoproliferative responses were markedly