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Publikováno v:
PROTEOMICS. 11:3657-3664
We previously used proteomics technology to globally identify gastric cancer-associated proteins and found that gastrokine 1 (GKN1) was dramatically underexpressed in gastric cancer tissues. Here, we further showed that GKN1 could inhibit cell growth
Autor:
Patrick Lécine, Subhash Thalappilly, Philippe Soubeyran, Odile Gayet, Muhtadi Suliman, Juan L. Iovanna, Aurélie Hermant, Nelson Dusetti
Publikováno v:
PROTEOMICS. 8:3071-3081
Pancreatic ductal adenocarcinoma (PDAC) is a fatal disease that shows minimal response to chemotherapy. Genetic changes involved in the progression of PDAC concern genes that encode proteins related to signal transduction networks. This fact reveals
Autor:
Qingsong Lin, Ping Chen, Canhua Huang, Quek Choon Lau, Jiong Li, Aiping Tong, Lijuan Chen, Yuquan Wei, Xia Zhao, Lihong Wu, Hong Tang
Publikováno v:
PROTEOMICS. 8:2012-2023
Hepatitis B virus (HBV) is one of the major etiological factors responsible for acute and chronic liver disease and for the development of hepatocellular carcinoma (HCC). To determine the effects of HBV replication on host cell-protein expression, we
Autor:
Rosamonde E. Banks, Sarah Hanrahan, Nick Totty, Rainy Mears, Peter Selby, Poulam M. Patel, Sarah L. Young, Carol Upton, Rachel A. Craven
Publikováno v:
PROTEOMICS. 4:4019-4031
Exosomes are 40-100 nm vesicles released by numerous cell types and are thought to have a variety of roles depending on their origin. Exosomes derived from antigen presenting cells have been shown to be capable of initiating immune responses in vivo
Publikováno v:
Proteomics. 9(3)
Exposure of glioblastoma U87MG cells to a cAMP analog leads to a decrease in proliferation, invasion, and angiogenic potential. Here, we apply a label-free MS-based approach to identify formerly N-linked glycopeptides that change in abundance upon cA
Autor:
Alberto Bavelloni, Irene Faenza, Lucio Cocco, Daniela Parisi, Manuela Piazzi, Nadir M. Maraldi, Ivan Matic, Gabriella Cioffi
Publikováno v:
Proteomics
An extensive body of evidence links inositide-specific phospholipase C (PLC) to the nucleus and the main isoform located in the nucleus is PLCbeta(1). Constitutive overexpression of nuclear PLCbeta(1) has been previously shown to inhibit Friend eryth