Zobrazeno 1 - 10
of 41
pro vyhledávání: '"35"'
Autor:
Xiu-Fen Bu, Jia-Ming Zhang, Wei-Xin Hu, Jiang Li, Saiqun Luo, De-Hui Xiong, Yali Wang, Guangdi Li, Jingping Hu
Publikováno v:
Oncogene. 39:3354-3366
Multiple myeloma (MM) is a clinically and biologically heterogenous event that accounts for approximately 10% of all hematological malignancies. Chromosome 1 open reading frame 35 (C1orf35) is a gene cloned and identified in our laboratory from a MM
Publikováno v:
Oncogene. 18:4968-4973
Tpl-2/Cot proto-oncogene encodes a serine threonine kinase and was initially cloned as a provirus insertion site in MoMuLV-induced T cell lymphomas in rats. Tpl-2 locus was also shown to be affected by provirus insertion in MMTV-induced mammary carci
Autor:
Kim Wright, Igor Filippovich, Georgia Chenevix-Trench, Geoff W. Birrell, Evgeny N. Imyanitov, Kum Kum Khanna, Michael Walsh, Jeremy Arnold, Martin F. Lavin, Michelle A. Mould, Natasha Sorokina, Samuel C. Mok
Publikováno v:
Oncogene. 18:4640-4642
Loss of heterozygosity (LOH) involving the distal part of the short arm of chromosome 1 occurs frequently in ovarian adenocarcinomas but the tumour suppressor gene(s) targeted by this event is unknown. We have used five microsatellite markers in a pa
Autor:
Mike Hubank, Magali Williamson, Simon Bott, Atique U. Ahmed, Q Wang, Alex Freeman, Nicola Brookman-Amissah, Joseph Nariculam, John R. W. Masters
Publikováno v:
Oncogene. 26(45)
Oligoarray analysis of a matched pair of prostate cancer and normal cell lines derived from the same radical prostatectomy specimen identified 113 candidate hypomethylated genes that were overexpressed in the cancer cells and contained CpG islands. H
Autor:
Marta Monteiro, Béatrice Loriod, Olivier Delattre, Eugene Novikov, Isabelle Janoueix-Lerosey, Catherine Nguyen, Nadège Gruel, Gudrun Schleiermacher
Publikováno v:
Oncogene
Oncogene, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. 〈10.1038/sj.onc.1207784〉
Oncogene, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. 〈10.1038/sj.onc.1207784〉
Deletion of the chromosome 1p36 region is a frequent abnormality in neuroblastoma. To gain further insights into the role of this alteration in oncogenesis, we have constructed a specific cDNA microarray representing most known genes and ESTs from th
Autor:
Guiji Zhang, Xiaoyuan Li, Yaqiu Zheng, Kai Wang, Xia Tang, Wanfeng Zhang, Dewei Li, Bingtao Hao, Dachun Zhao, Keyue Ding, Ni Tang, Yanhong Chen, Li Liang
Publikováno v:
Oncogene. 39(16)
Liver hepatocellular carcinoma (LIHC) is the second leading cause of cancer mortality worldwide. Although cancer driver genes identified so far have been considered to be saturated or nearly saturated, challenges remain in discovering novel genes und
Autor:
D. Dumas, A. Diot, N. de Freitas Caires, Michael W.H. Coughtrie, Isabelle Bertin-Jung, A. M. Thompson, Xianqing Mao, Sandrine Gulberti, Caroline Gauche, F. Merhi-Soussi, J-C Bourdon, Catherine Bui, Mohamed Ouzzine, Sylvie Fournel-Gigleux, Nick Ramalanjaona
Publikováno v:
Oncogene
Oncogene, Nature Publishing Group, 2016, 35 (38), pp.5043-5055. ⟨10.1038/onc.2016.44⟩
Oncogene, Nature Publishing Group, 2016, 35 (38), pp.5043-5055. ⟨10.1038/onc.2016.44⟩
International audience; Heparan sulfate (HS) proteoglycan chains are key components of the breast tumor microenvironment that critically influence the behavior of cancer cells. It is established that abnormal synthesis and processing of HS play a pro
Autor:
Giulia Piaggio, Dawid Walerych, Daniela Trisciuoglio, Aymone Gurtner, Teresa Colombo, G Del Sal, Kamil Lisek, Gianluca Bossi, F Garibaldi, Paola Paci, Emmanuela Falcone
Publikováno v:
Oncogene (Basingstoke, Online) 35 (2016): 3760–3770.
info:cnr-pdr/source/autori:Aymone Gurtner, Francesca Garibaldi, Emmanuela Falcone, Daniela Trisciuoglio, Teresa Colombo, Kamil Lisek, Dawid Walerych, Giannino Dal Sal, Paola Paci, Gianluca Bossi, and Giulia Piaggio/titolo:Mutant p53 inhibits miRNA biogenesis by interfering with the Microprocessor complex/doi:/rivista:Oncogene (Basingstoke, Online)/anno:2016/pagina_da:3760/pagina_a:3770/intervallo_pagine:3760–3770/volume:35
info:cnr-pdr/source/autori:Aymone Gurtner, Francesca Garibaldi, Emmanuela Falcone, Daniela Trisciuoglio, Teresa Colombo, Kamil Lisek, Dawid Walerych, Giannino Dal Sal, Paola Paci, Gianluca Bossi, and Giulia Piaggio/titolo:Mutant p53 inhibits miRNA biogenesis by interfering with the Microprocessor complex/doi:/rivista:Oncogene (Basingstoke, Online)/anno:2016/pagina_da:3760/pagina_a:3770/intervallo_pagine:3760–3770/volume:35
Downregulation of microRNAs (miRNAs) is commonly observed in cancers and promotes tumorigenesis suggesting that miRNAs may function as tumor suppressors. However, the mechanism through which miRNAs are regulated in cancer, and the connection between
Autor:
Malay Haldar, Michael J. Monument, Richard K. Wilson, Jared J. Barrott, R. L. Randall, Mario R. Capecchi, Kevin B. Jones, Mingchao Xie, Ellen M. Langer, Timothy L. Mosbruger, Ju Fen Zhu, Huifeng Jin, Li Ding, Bradley R. Cairns
Publikováno v:
Oncogene
Oncogene, vol 35, iss 38
Oncogene, vol 35, iss 38
Synovial sarcomas are aggressive soft-tissue malignancies that express chromosomal translocation-generated fusion genes, SS18-SSX1 or SS18-SSX2 in most cases. Here, we report a mouse sarcoma model expressing SS18-SSX1, complementing our prior model e
Autor:
Jenny Joutsen, Fang Cheng, Mikael C. Puustinen, Johanna K. Björk, Lea Sistonen, Malin Åkerfelt, Matthias Nees
Publikováno v:
Björk, J K, Åkerfelt, M, Joutsen, J, Puustinen, M C, Cheng, F, Sistonen, L & Nees, M 2016, ' Heat-shock factor 2 is a suppressor of prostate cancer invasion ', Oncogene, vol. 35, no. 14, pp. 1770-84 . https://doi.org/10.1038/onc.2015.241
Oncogene
Oncogene
Heat-shock factors (HSFs) are key transcriptional regulators in cell survival. Although HSF1 has been identified as a driver of carcinogenesis, HSF2 has not been explored in malignancies. Here, we report that HSF2 suppresses tumor invasion of prostat