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pro vyhledávání: '"35"'
Publikováno v:
Oncogene. 18:4968-4973
Tpl-2/Cot proto-oncogene encodes a serine threonine kinase and was initially cloned as a provirus insertion site in MoMuLV-induced T cell lymphomas in rats. Tpl-2 locus was also shown to be affected by provirus insertion in MMTV-induced mammary carci
Autor:
Marta Monteiro, Béatrice Loriod, Olivier Delattre, Eugene Novikov, Isabelle Janoueix-Lerosey, Catherine Nguyen, Nadège Gruel, Gudrun Schleiermacher
Publikováno v:
Oncogene
Oncogene, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. 〈10.1038/sj.onc.1207784〉
Oncogene, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. ⟨10.1038/sj.onc.1207784⟩
Oncogene, Nature Publishing Group, 2004, 23 (35), pp.5912-5922. 〈10.1038/sj.onc.1207784〉
Deletion of the chromosome 1p36 region is a frequent abnormality in neuroblastoma. To gain further insights into the role of this alteration in oncogenesis, we have constructed a specific cDNA microarray representing most known genes and ESTs from th
Autor:
Khalil Arar, Christina Laurini, Michèle Sabbah, Laurent Vallar, Michèle Moes, Charles-Henri Lecellier, Evelyne Friederich, A. Le Bechec, Guillaume Vetter, Anne Saumet, Charles Theillet
Publikováno v:
Oncogene
Oncogene, 2010, 29 (31), pp.4436-48. ⟨10.1038/onc.2010.181⟩
Oncogene, Nature Publishing Group, 2010, 29 (31), pp.4436-48. ⟨10.1038/onc.2010.181⟩
Oncogene, 35(5), 670. Nature Publishing Group (2016).
Oncogene, Nature Publishing Group, 2010, 29 (31), pp.4436-48. 〈10.1038/onc.2010.181〉
Oncogene, 2010, 29 (31), pp.4436-48. ⟨10.1038/onc.2010.181⟩
Oncogene, Nature Publishing Group, 2010, 29 (31), pp.4436-48. ⟨10.1038/onc.2010.181⟩
Oncogene, 35(5), 670. Nature Publishing Group (2016).
Oncogene, Nature Publishing Group, 2010, 29 (31), pp.4436-48. 〈10.1038/onc.2010.181〉
4; International audience; Epithelial to mesenchymal transition (EMT) is a key step toward metastasis. MCF7 breast cancer cells conditionally expressing the EMT master regulator SNAI1 were used to identify early expressed microRNAs (miRNAs) and their
Publikováno v:
Oncogene. 25:1391-1399
We have previously shown that the breast cancer susceptibility gene, BRCA1, can transcriptionally activate the p27(Kip1) promoter. The BRCA1-responsive element was defined as a 35 bp region from position -545 to -511. We next determined that within t
Autor:
Teresa Druck, Ming You, Dimitrios Iliopoulos, Gulnur Guler, Michael A. Pereira, Clinton J. Grubbs, Zhongqiu Zhang, Ronald A. Lubet, Louise Y.Y. Fong, Kay Huebner, Shuang Yin Han
Publikováno v:
Oncogene. 23(22)
To determine if: (1) 5' CpG island methylation is related to Fhit inactivation; (2) there are tumor or carcinogen-specific methylation patterns, we examined 35 CpG sites in the promoter, exon and intron 1 of the mouse Fhit gene. In primary tumors of
Autor:
Ralph B. Arlinghaus, Feng Lin, Jiaxin Liu, Giuseppe Monaco, Hui Lin, Tong Sun, Clifton Stephens, John W. Belmont
Publikováno v:
Oncogene. 20(15)
It is well accepted that the Bcr-Abl oncoprotein encoded by the Philadelphia chromosome is responsible for causing chronic myelogenous leukemia (CML). We have previously demonstrated that expression of Bcr interferes with the oncogenic effects of Bcr