Zobrazeno 1 - 2
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pro vyhledávání: '"Sander Barnhoorn"'
Autor:
Annelieke S. de Wit, Sander Barnhoorn, Mirjam M.C. Wamelink, Renata M. C. Brandt, César Payán-Gómez, Akos Gyenis, Jan H.J. Hoeijmakers, Wilbert P. Vermeij, Pier G. Mastroberardino, André B.P. van Kuilenburg, Donatella Caruso, Sara Sepe, Matteo Audano, René Leen, Roel C. Janssens, Nico Mitro, Cíntia R. Bombardieri, Chiara Milanese, Silvia Pedretti
Publikováno v:
Nature Communications, 10:4887. Nature Publishing Group
Nature Communications
Repositorio EdocUR-U. Rosario
Universidad del Rosario
instacron:Universidad del Rosario
Nature Communications, Vol 10, Iss 1, Pp 1-16 (2019)
Nature communications, 10(1):4887. Nature Publishing Group
Nature Communications, 10(1):4887. Nature Publishing Group
Milanese, C, Bombardieri, C N R, Sepe, S, Barnhoorn, S, Payán-Goméz, C S, Caruso, D, Audano, M, Pedretti, S, Vermeij, W P, Brandt, R M C, Gyenis, A, Wamelink, M M, de Wit, A S, Janssens, R C, Leen, R, van Kuilenburg, A B P, Mitro, N, Hoeijmakers, J H J & Mastroberardino, P G 2019, ' DNA damage and transcription stress cause ATP-mediated redesign of metabolism and potentiation of anti-oxidant buffering ', Nature Communications, vol. 10, no. 1, 4887 . https://doi.org/10.1038/s41467-019-12640-5
Nature Communications
Repositorio EdocUR-U. Rosario
Universidad del Rosario
instacron:Universidad del Rosario
Nature Communications, Vol 10, Iss 1, Pp 1-16 (2019)
Nature communications, 10(1):4887. Nature Publishing Group
Nature Communications, 10(1):4887. Nature Publishing Group
Milanese, C, Bombardieri, C N R, Sepe, S, Barnhoorn, S, Payán-Goméz, C S, Caruso, D, Audano, M, Pedretti, S, Vermeij, W P, Brandt, R M C, Gyenis, A, Wamelink, M M, de Wit, A S, Janssens, R C, Leen, R, van Kuilenburg, A B P, Mitro, N, Hoeijmakers, J H J & Mastroberardino, P G 2019, ' DNA damage and transcription stress cause ATP-mediated redesign of metabolism and potentiation of anti-oxidant buffering ', Nature Communications, vol. 10, no. 1, 4887 . https://doi.org/10.1038/s41467-019-12640-5
Accumulation of DNA lesions causing transcription stress is associated with natural and accelerated aging and culminates with profound metabolic alterations. Our understanding of the mechanisms governing metabolic redesign upon genomic instability, h
Publikováno v:
Nature Communications