Zobrazeno 1 - 10
of 14
pro vyhledávání: '"133"'
Reduced Il17a Expression Distinguishes a Ly6cloMHCIIhi Macrophage Population Promoting Wound Healing
Autor:
Mathieu P Rodero, Christophe Combadière, Samantha Hodgson, Brett G. Hollier, Kiarash Khosrotehrani
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Macrophages are the main components of inflammation during skin wound healing. They are critical in wound closure and in excessive inflammation, resulting in defective healing observed in chronic wounds. Given the heterogeneity of macrophage phenotyp
Autor:
Jeffrey H. Dunn, David A. Norris, Mayumi Fujita, Weimin Liu, Charles A. Dinarello, Yuchun Luo
Publikováno v:
The Journal of investigative dermatology
Journal of Investigative Dermatology, 133, 518-27
Journal of Investigative Dermatology, 133, 2, pp. 518-27
Journal of Investigative Dermatology, 133, 518-27
Journal of Investigative Dermatology, 133, 2, pp. 518-27
Item does not contain fulltext Apoptosis-associated Speck-like protein containing a CARD (caspase recruitment domain) (ASC) was originally named because it triggered apoptosis in certain tumors. More recently, however, ASC was found to be a central a
Autor:
Ulrike Lichti, Andrew Ryscavage, Christophe Cataisson, Stuart H. Yuspa, Dolph L. Hatfield, Marcus Conrad, Susan M. Fischer, Bradley A. Carlson, Carol Mikulec, Aniruddha Sengupta
Publikováno v:
The journal of investigative dermatology 133(7), 1731-1741 (2013). doi:10.1038/jid.2013.52
J. Invest. Dermatol. 133, 1731-1741 (2013)
J. Invest. Dermatol. 133, 1731-1741 (2013)
Selenoproteins are essential molecules for the mammalian antioxidant network. We previously demonstrated that targeted loss of all selenoproteins in mouse epidermis disrupted skin and hair development, and caused premature death. In the current study
Autor:
Melanie Hupe, Kyungho Park, Peter M. Elias, Richard L. Gallo, Walter M. Holleran, Andrew W. Borkowski, Kyong-Oh Shin, Yong-Moon Lee, Yoshikazu Uchida
Publikováno v:
The Journal of investigative dermatology, vol 133, iss 8
Park, K; Elias, PM; Hupe, M; Borkowski, AW; Gallo, RL; Shin, KO; et al.(2013). Resveratrol stimulates sphingosine-1-phosphate signaling of cathelicidin production. Journal of Investigative Dermatology, 133(8), 1942-1949. doi: 10.1038/jid.2013.133. UCSF: Retrieved from: http://www.escholarship.org/uc/item/95t0g21r
The Journal of investigative dermatology
Park, K; Elias, PM; Hupe, M; Borkowski, AW; Gallo, RL; Shin, KO; et al.(2013). Resveratrol stimulates sphingosine-1-phosphate signaling of cathelicidin production. Journal of Investigative Dermatology, 133(8), 1942-1949. doi: 10.1038/jid.2013.133. UCSF: Retrieved from: http://www.escholarship.org/uc/item/95t0g21r
The Journal of investigative dermatology
We recently discovered a regulatory mechanism that stimulates the production of the multifunctional antimicrobial peptide cathelicidin antimicrobial peptide (CAMP). In response to subtoxic levels of ER stress, increased sphingosine-1-phosphate (S1P)
Autor:
Mathieu P Rodero, Herlina Y. Handoko, Graeme J. Walker, Geoffrey R. Hill, Blake Ferguson, Glen M. Boyle, Christian R. Engwerda, Kiarash Khosrotehrani, H. Konrad Muller
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (7), pp.1803-1812. ⟨10.1038/JID.2013.9⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (7), pp.1803-1812. ⟨10.1038/JID.2013.9⟩
Intermittent sunburns, particularly in childhood, are the strongest environmental risk factor for malignant melanoma (MM). In mice, a single neonatal UVR exposure induces MM, whereas chronic doses to adult mice do not. Neonatal UVR alters melanocyte
Autor:
Martin Meuli, Ossia M. Eichhoff, Ernst Reichmann, Sophie Böttcher-Haberzeth, Erik Braziulis, Daniel S. Widmer, Luca Pontiggia, Clemens Schiestl, Claudia Meuli-Simmen, Thomas Biedermann, Bart Hendriks
Publikováno v:
Journal of Investigative Dermatology; Vol 133
Recently, Biedermann et al. (2010) have demonstrated that human eccrine sweat gland cells can develop a multilayered epidermis. The question still remains whether these cells can fulfill exclusive and very specific functional properties of epidermal
Autor:
Zenzo Isogai, Seiya Matsui, Yosuke Yagi, Yoshiki Miyachi, Eri Muroga, Susumu Ikehara, Atsushi Utani, Shigehiko Suzuki, Motoko Naitoh
Publikováno v:
Journal of Investigative Dermatology. 133:386-393
The most distinctive feature of keloid is the extreme deposition of extracellular matrix, including collagens and proteoglycans (PGs). The focus of this study was the PG versican, which presumably defines keloid volume because of its ability to retai
Autor:
Martin Hrabé de Angelis, Helmut Fuchs, Jenny Li-Ying Huang, Mugdha Deo, Catherine D. Van Raamsdonk
Publikováno v:
J. Invest. Dermatol. 133, 49-58 (2013)
Mutations in neurofibromin (NF1) cause the dominant genetic disorder neurofibromatosis type 1. Neurofibromatosis is characterized by Schwann cell-based tumors and skin hyperpigmentation, resulting from both haploinsufficiency and loss of heterozygosi
Autor:
Hiroshi Shimizu, Gang Wang, Yasuyuki Fujita, Michitaka Ozaki, Nao Saito, Sanae Haga, Hongjiang Qiao, Yoshihiro Ohmiya, Riichiro Abe, Inkin Hayashi-Ujiie, Chun Wu
Publikováno v:
Journal of Investigative Dermatology. 133:841-843
Correction to: Journal of Investigative Dermatology (2013) 133, 841–843; doi:10.1038/jid.2012.345; published online 27 September 2012
Autor:
Rita Haapakoski, Terhi Savinko, Henrik Wolff, Harri Alenius, Antti Lauerma, Nanna Fyhrquist, Sari Lehtimäki, Piia Karisola
Publikováno v:
Journal of Investigative Dermatology; Vol 133
Toll-like receptors (TLRs) are pattern-recognition receptors that have a pivotal role as primary sensors of microbial products and as initiators of innate and adaptive immune responses. We investigated the role of TLR2, TLR3, and TLR4 activation duri