Zobrazeno 1 - 8
of 8
pro vyhledávání: '"133"'
Autor:
Pilar Galan, Anne Bernard, Toufik Labib, Jean-François Zagury, Cédric Coulonges, Julie Latreille, Wassila Carpentier, R. Jdid, Olivier Delaneau, Lieng Taing, Erwin Tschachler, Serge Hercberg, Frédérique Morizot, Christiane Guinot, Denis Malvy, Sigrid Le Clerc, Safa Melak, Khaled Ezzedine
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
Journal of Investigative Dermatology, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
Journal of Investigative Dermatology, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
International audience; A genome-wide association study (GWAS) was conducted on 502 French middle-aged Caucasian women to identify genetic factors that may affect skin aging severity. A high-throughput Illumina Human Omni1-Quad beadchip was used. Aft
Reduced Il17a Expression Distinguishes a Ly6cloMHCIIhi Macrophage Population Promoting Wound Healing
Autor:
Mathieu P Rodero, Christophe Combadière, Samantha Hodgson, Brett G. Hollier, Kiarash Khosrotehrani
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Macrophages are the main components of inflammation during skin wound healing. They are critical in wound closure and in excessive inflammation, resulting in defective healing observed in chronic wounds. Given the heterogeneity of macrophage phenotyp
Autor:
Jeffrey H. Dunn, David A. Norris, Mayumi Fujita, Weimin Liu, Charles A. Dinarello, Yuchun Luo
Publikováno v:
The Journal of investigative dermatology
Journal of Investigative Dermatology, 133, 518-27
Journal of Investigative Dermatology, 133, 2, pp. 518-27
Journal of Investigative Dermatology, 133, 518-27
Journal of Investigative Dermatology, 133, 2, pp. 518-27
Item does not contain fulltext Apoptosis-associated Speck-like protein containing a CARD (caspase recruitment domain) (ASC) was originally named because it triggered apoptosis in certain tumors. More recently, however, ASC was found to be a central a
Autor:
Yonghu Sun, Hong Liu, Jing Cao, Wenjun Yu, Furen Zhang, Fangfang Bao, Zhenhua Yue, Zhenzhen Wang, Chuan Wang
Publikováno v:
Journal of Investigative Dermatology. 138:1101-1106
Dapsone hypersensitivity syndrome is a rare yet severe adverse drug reaction caused by dapsone, a principal drug in multidrug therapy for leprosy. HLA-B*13:01 has been identified as a strong risk factor of dapsone hypersensitivity syndrome; however,
Autor:
Melanie Hupe, Kyungho Park, Peter M. Elias, Richard L. Gallo, Walter M. Holleran, Andrew W. Borkowski, Kyong-Oh Shin, Yong-Moon Lee, Yoshikazu Uchida
Publikováno v:
The Journal of investigative dermatology, vol 133, iss 8
Park, K; Elias, PM; Hupe, M; Borkowski, AW; Gallo, RL; Shin, KO; et al.(2013). Resveratrol stimulates sphingosine-1-phosphate signaling of cathelicidin production. Journal of Investigative Dermatology, 133(8), 1942-1949. doi: 10.1038/jid.2013.133. UCSF: Retrieved from: http://www.escholarship.org/uc/item/95t0g21r
The Journal of investigative dermatology
Park, K; Elias, PM; Hupe, M; Borkowski, AW; Gallo, RL; Shin, KO; et al.(2013). Resveratrol stimulates sphingosine-1-phosphate signaling of cathelicidin production. Journal of Investigative Dermatology, 133(8), 1942-1949. doi: 10.1038/jid.2013.133. UCSF: Retrieved from: http://www.escholarship.org/uc/item/95t0g21r
The Journal of investigative dermatology
We recently discovered a regulatory mechanism that stimulates the production of the multifunctional antimicrobial peptide cathelicidin antimicrobial peptide (CAMP). In response to subtoxic levels of ER stress, increased sphingosine-1-phosphate (S1P)
Autor:
Martin Meuli, Ossia M. Eichhoff, Ernst Reichmann, Sophie Böttcher-Haberzeth, Erik Braziulis, Daniel S. Widmer, Luca Pontiggia, Clemens Schiestl, Claudia Meuli-Simmen, Thomas Biedermann, Bart Hendriks
Publikováno v:
Journal of Investigative Dermatology; Vol 133
Recently, Biedermann et al. (2010) have demonstrated that human eccrine sweat gland cells can develop a multilayered epidermis. The question still remains whether these cells can fulfill exclusive and very specific functional properties of epidermal
Autor:
Martin Hrabé de Angelis, Helmut Fuchs, Jenny Li-Ying Huang, Mugdha Deo, Catherine D. Van Raamsdonk
Publikováno v:
J. Invest. Dermatol. 133, 49-58 (2013)
Mutations in neurofibromin (NF1) cause the dominant genetic disorder neurofibromatosis type 1. Neurofibromatosis is characterized by Schwann cell-based tumors and skin hyperpigmentation, resulting from both haploinsufficiency and loss of heterozygosi
Autor:
Rita Haapakoski, Terhi Savinko, Henrik Wolff, Harri Alenius, Antti Lauerma, Nanna Fyhrquist, Sari Lehtimäki, Piia Karisola
Publikováno v:
Journal of Investigative Dermatology; Vol 133
Toll-like receptors (TLRs) are pattern-recognition receptors that have a pivotal role as primary sensors of microbial products and as initiators of innate and adaptive immune responses. We investigated the role of TLR2, TLR3, and TLR4 activation duri