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Reduced Il17a Expression Distinguishes a Ly6cloMHCIIhi Macrophage Population Promoting Wound Healing
Autor:
Mathieu P Rodero, Christophe Combadière, Samantha Hodgson, Brett G. Hollier, Kiarash Khosrotehrani
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Macrophages are the main components of inflammation during skin wound healing. They are critical in wound closure and in excessive inflammation, resulting in defective healing observed in chronic wounds. Given the heterogeneity of macrophage phenotyp
Autor:
Martin Meuli, Ossia M. Eichhoff, Ernst Reichmann, Sophie Böttcher-Haberzeth, Erik Braziulis, Daniel S. Widmer, Luca Pontiggia, Clemens Schiestl, Claudia Meuli-Simmen, Thomas Biedermann, Bart Hendriks
Publikováno v:
Journal of Investigative Dermatology; Vol 133
Recently, Biedermann et al. (2010) have demonstrated that human eccrine sweat gland cells can develop a multilayered epidermis. The question still remains whether these cells can fulfill exclusive and very specific functional properties of epidermal
Autor:
Martin Hrabé de Angelis, Helmut Fuchs, Jenny Li-Ying Huang, Mugdha Deo, Catherine D. Van Raamsdonk
Publikováno v:
J. Invest. Dermatol. 133, 49-58 (2013)
Mutations in neurofibromin (NF1) cause the dominant genetic disorder neurofibromatosis type 1. Neurofibromatosis is characterized by Schwann cell-based tumors and skin hyperpigmentation, resulting from both haploinsufficiency and loss of heterozygosi