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pro vyhledávání: '"133"'
Publikováno v:
Journal of Investigative Dermatology. 141:S171
Autor:
J. Newman, Larisa J. Geskin, C. Del Guzzo, Yeun-Hee Anna Park, A. Levin, A. Dana, E. Langhoff, Ravi Vinnakota
Publikováno v:
Journal of Investigative Dermatology. 136:S24
Autor:
Pilar Galan, Anne Bernard, Toufik Labib, Jean-François Zagury, Cédric Coulonges, Julie Latreille, Wassila Carpentier, R. Jdid, Olivier Delaneau, Lieng Taing, Erwin Tschachler, Serge Hercberg, Frédérique Morizot, Christiane Guinot, Denis Malvy, Sigrid Le Clerc, Safa Melak, Khaled Ezzedine
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
Journal of Investigative Dermatology, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
Journal of Investigative Dermatology, 2013, 133 (4), pp.929-935. ⟨10.1038/jid.2012.458⟩
International audience; A genome-wide association study (GWAS) was conducted on 502 French middle-aged Caucasian women to identify genetic factors that may affect skin aging severity. A high-throughput Illumina Human Omni1-Quad beadchip was used. Aft
Autor:
Joyce Y. Tung, Axel M. Hillmer, Silke Redler, Vincent Mooser, Nicholas Eriksson, Dale R. Nyholt, Christine Herold, Amy K. Kiefer, Kari Stefansson, Nicholas G. Martin, Felix F. Brockschmidt, Stefanie Heilmann, J. Brent Richards, David A. Hinds, Ari Karason, Stavroula Kanoni, Nadine Fricker, Rui Li, George Dedoussis, Tim Becker, Sita H. Vermeulen, Regina C. Betz, Dmitriy Drichel, Markus M. Nöthen, Lambertus A. Kiemeney, Kijoung Song
Publikováno v:
Journal of Investigative Dermatology, 133, 6, pp. 1489-96
The journal of investigative dermatology 133(6), 1489-1496 (2013). doi:10.1038/jid.2013.43
Journal of Investigative Dermatology, 133, 1489-96
The journal of investigative dermatology 133(6), 1489-1496 (2013). doi:10.1038/jid.2013.43
Journal of Investigative Dermatology, 133, 1489-96
Item does not contain fulltext The pathogenesis of androgenetic alopecia (AGA, male-pattern baldness) is driven by androgens, and genetic predisposition is the major prerequisite. Candidate gene and genome-wide association studies have reported that
Reduced Il17a Expression Distinguishes a Ly6cloMHCIIhi Macrophage Population Promoting Wound Healing
Autor:
Mathieu P Rodero, Christophe Combadière, Samantha Hodgson, Brett G. Hollier, Kiarash Khosrotehrani
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Journal of Investigative Dermatology, 2013, 133 (3), pp.783-792. ⟨10.1038/JID.2012.368⟩
Macrophages are the main components of inflammation during skin wound healing. They are critical in wound closure and in excessive inflammation, resulting in defective healing observed in chronic wounds. Given the heterogeneity of macrophage phenotyp
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (2), pp.371--376. ⟨10.1038/jid.2012.279⟩
Journal of Investigative Dermatology, 2013, 133 (2), pp.371--376. ⟨10.1038/jid.2012.279⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (2), pp.371--376. ⟨10.1038/jid.2012.279⟩
Journal of Investigative Dermatology, 2013, 133 (2), pp.371--376. ⟨10.1038/jid.2012.279⟩
International audience; The primary outcome is a major component of a randomized clinical trial (RCT) and several types of outcome can be chosen in a given disease. We systematically reviewed RCTs on nonneoplastic dermatological diseases published in
Autor:
Yonghu Sun, Hong Liu, Jing Cao, Wenjun Yu, Furen Zhang, Fangfang Bao, Zhenhua Yue, Zhenzhen Wang, Chuan Wang
Publikováno v:
Journal of Investigative Dermatology. 138:1101-1106
Dapsone hypersensitivity syndrome is a rare yet severe adverse drug reaction caused by dapsone, a principal drug in multidrug therapy for leprosy. HLA-B*13:01 has been identified as a strong risk factor of dapsone hypersensitivity syndrome; however,
Autor:
Mathieu P Rodero, Herlina Y. Handoko, Graeme J. Walker, Geoffrey R. Hill, Blake Ferguson, Glen M. Boyle, Christian R. Engwerda, Kiarash Khosrotehrani, H. Konrad Muller
Publikováno v:
Journal of Investigative Dermatology
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (7), pp.1803-1812. ⟨10.1038/JID.2013.9⟩
Journal of Investigative Dermatology, Nature Publishing Group, 2013, 133 (7), pp.1803-1812. ⟨10.1038/JID.2013.9⟩
Intermittent sunburns, particularly in childhood, are the strongest environmental risk factor for malignant melanoma (MM). In mice, a single neonatal UVR exposure induces MM, whereas chronic doses to adult mice do not. Neonatal UVR alters melanocyte
Autor:
Martin Meuli, Ossia M. Eichhoff, Ernst Reichmann, Sophie Böttcher-Haberzeth, Erik Braziulis, Daniel S. Widmer, Luca Pontiggia, Clemens Schiestl, Claudia Meuli-Simmen, Thomas Biedermann, Bart Hendriks
Publikováno v:
Journal of Investigative Dermatology; Vol 133
Recently, Biedermann et al. (2010) have demonstrated that human eccrine sweat gland cells can develop a multilayered epidermis. The question still remains whether these cells can fulfill exclusive and very specific functional properties of epidermal
Autor:
Martin Hrabé de Angelis, Helmut Fuchs, Jenny Li-Ying Huang, Mugdha Deo, Catherine D. Van Raamsdonk
Publikováno v:
J. Invest. Dermatol. 133, 49-58 (2013)
Mutations in neurofibromin (NF1) cause the dominant genetic disorder neurofibromatosis type 1. Neurofibromatosis is characterized by Schwann cell-based tumors and skin hyperpigmentation, resulting from both haploinsufficiency and loss of heterozygosi