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Publikováno v:
Islets. 3:21-34
Diabetes (T1DM and T2DM) is characterized by a deficit in β-cell mass. A broader understanding of human β-cell replication mechanism is thus important to increase β-cell proliferation for future therapeutic interventions. Here, we show that p27 (K
Autor:
Melissa L. Brown, Amy S. Burnside, Alan L. Schneyer, Danielle Andrzejewski, Lara Bonomi, Nathan Ungerleider
Publikováno v:
Islets. 6:e1017226
Emerging evidence suggests that activin with its associated receptors, second messengers, and antagonists would be excellent targets for therapeutic drug development in the treatment of diabetes. We undertook the current study to investigate the abil