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Autor:
Giovannino Silvestri, Pavel Burda, Pavla Pecherkova, Rossana Trotta, Klara Srutova, Filipp Savvulidi, Zofie Sovova, Hana Klamova, Tomas Stopka, Katerina Machova Polakova, Danilo Perrotti, Jitka Koblihova, Nikola Curik
Publikováno v:
Haematologica
The fusion oncoprotein BCR-ABL1 exhibits aberrant tyrosine kinase activity and it has been proposed that it deregulates signaling networks involving both transcription factors and non-coding microRNAs that result in chronic myeloid leukemia (CML). Pr