Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Hossein Ardeschir Ghofrani"'
Publikováno v:
European Respiratory Review, Vol 23, Iss 134, Pp 469-475 (2014)
Pulmonary arterial hypertension (PAH) is a complex, progressive disease with several pathobiological mechanisms, including the endothelin, nitric oxide and prostacyclin pathways. Current treatments for PAH target one of these pathways and, in more se
Publikováno v:
European Respiratory Review, Vol 26, Iss 143 (2017)
Chronic thromboembolic pulmonary hypertension (CTEPH) results from incomplete resolution of acute pulmonary emboli, organised into fibrotic material that obstructs large pulmonary arteries, and distal small-vessel arteriopathy. Pulmonary endarterecto
Autor:
Philippe Brenot, Hiromi Matsubara, Irene Lang, Takeshi Ogo, Bernhard C. Meyer, Eckhard Mayer, Hossein Ardeschir Ghofrani, Marcin Kurzyna
Publikováno v:
European Respiratory Review, Vol 26, Iss 143 (2017)
Chronic thromboembolic pulmonary hypertension (CTEPH) is thought to result from incomplete resolution of pulmonary thromboemboli that undergo organisation into fibrous tissue within pulmonary arterial branches, filling pulmonary arterial lumina with
Autor:
R. T. Schermuly, Stefan Schäfer, F. Grimminger, Hossein Ardeschir Ghofrani, Hendrik Milting, Bernd Riedl, Martina Klein, Peter Ellinghaus
Publikováno v:
European Respiratory Review, Vol 17, Iss 108, Pp 72-73 (2008)
Inhibition of tyrosine kinases can reverse pulmonary hypertension but little is known about the role of serine-/threonine kinases in vascular and myocardial remodeling. We investigated the effects of sorafenib, an inhibitor of the tyrosine kinases VE
Publikováno v:
European Respiratory Review, Vol 23, Iss 134, Pp 469-475 (2014)
Pulmonary arterial hypertension (PAH) is a complex, progressive disease with several pathobiological mechanisms, including the endothelin, nitric oxide and prostacyclin pathways. Current treatments for PAH target one of these pathways and, in more se
Externí odkaz:
https://doaj.org/article/7b52e25d8b7d4087bb32e96a7d1dc752
Autor:
Irene Lang, Bernhard C. Meyer, Takeshi Ogo, Hiromi Matsubara, Marcin Kurzyna, Hossein-Ardeschir Ghofrani, Eckhard Mayer, Philippe Brenot
Publikováno v:
European Respiratory Review, Vol 26, Iss 143 (2017)
Chronic thromboembolic pulmonary hypertension (CTEPH) is thought to result from incomplete resolution of pulmonary thromboemboli that undergo organisation into fibrous tissue within pulmonary arterial branches, filling pulmonary arterial lumina with
Externí odkaz:
https://doaj.org/article/d6c2a03993844923b8e64aa647d26226