Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Wojciech Wojakowski"'
Autor:
Anna Błach, Andrzej Ochała, Łukasz Rzeszutko, Łukasz Partyka, Wojciech Rychlik, Monika Dzier˙zak-Mietła, Wieslaw Cybulski, Magdalena Kostkiewicz, Wojciech Wańha, Witold Włudarczyk, Tomasz Jadczyk, Katarzyna Gruszczyńska, Miroslaw Markiewicz, Michal Tendera, Grzegorz Smolka, Beata Ochala, Aleksandra Michalewska-Włudarczyk, Tomasz Pawłowski, Sebastian Dworowy, Zofia Parma, Joanna Ciosek, Wojciech Zasada, Wojciech Wojakowski, Wacław Kuczmik
Publikováno v:
Circulation Research. 120:670-680
Rationale : New therapies for refractory angina are needed. Objective : Assessment of transendocardial delivery of bone marrow CD133 + cells in patients with refractory angina. Methods and Results : Randomized, double-blinded, placebo-controlled tria
Autor:
Mariann, Gyöngyösi, Wojciech, Wojakowski, Eliano P, Navarese, Lemuel À, Moye, Robert D, Simari
Publikováno v:
Circulation research. 118(8)
In contrast to multiple publication-based meta-analyses involving clinical cardiac regeneration therapy in patients with recent myocardial infarction, a recently published meta-analysis based on individual patient data reported no effect of cell ther
Autor:
Kai Zacharowski, Jean-Loup Bascands, Andrew Herman, Wojciech Wojakowski, Patrick E. MacDonald, Gianni D Angelini, Nicolle Kränkel, Paola Campagnolo, Lucíola S. Barcelos, Paolo Madeddu, Jocelyn E. Manning Fox, Rajesh Katare, Mauro Siragusa, Raimondo Ascione, Nguyen Tran, Iwona Mroz, Costanza Emanueli, Joost P. Schanstra, Orazio Fortunato, Giuseppe Mangialardi, Gaia Spinetti
Publikováno v:
Circulation Research
Circulation Research, American Heart Association, 2008, 103 (11), pp.1335-43. ⟨10.1161/CIRCRESAHA.108.179952⟩
Circulation Research, 2008, 103 (11), pp.1335-43. ⟨10.1161/CIRCRESAHA.108.179952⟩
Circulation Research, American Heart Association, 2008, 103 (11), pp.1335-43. ⟨10.1161/CIRCRESAHA.108.179952⟩
Circulation Research, 2008, 103 (11), pp.1335-43. ⟨10.1161/CIRCRESAHA.108.179952⟩
Reduced migratory function of circulating angiogenic progenitor cells (CPCs) has been associated with impaired neovascularization in patients with cardiovascular disease (CVD). Previous findings underline the role of the kallikrein-kinin system in an