Zobrazeno 1 - 10
of 1 329
pro vyhledávání: ''
Autor:
Naohito Nishii, Mifumi Kawabe, Minami Goto, Hiroki Sakai, Kyoko Yoshizaki, Takashi Mori, Akihiro Hirata
Publikováno v:
Carcinogenesis. 42:70-79
Many hereditary disorders in dogs have equivalents in humans and thus attract attention as natural animal models. Breed predisposition to certain diseases often provides promising clues to explore novel hereditary disorders in dogs. Recently, cases o
Autor:
Yu You, Dai-Wen Su, Zhi-Qing Zhang, Yan Liu, Wei Zhu, Zhenru Wu, Jian Hu, Yun-Bing Wang, Jianping Gong
Publikováno v:
Carcinogenesis. 41:1576-1582
The difference of the patients bearing hepatocellular carcinoma (HCC) with and without cirrhosis at clinical level has not been completely determined. This study compared their differences in clinicopathological traits and prognostic factors for rela
Publikováno v:
Carcinogenesis. 41:909-917
Imbalance of the gut microbial community promotes inflammation and colorectal cancer (CRC). Previously, we demonstrated that freeze-dried Parabacteroides distasonis (Pd) suppressed obesity-driven colorectal tumorigenesis in mice. Here, we investigate
Autor:
Caroline R. Delma, Samy L. Habib, Krishnan Manickam, Sumathy Mohan, Mohan Natarajan, Sureshkumar M A
Publikováno v:
Carcinogenesis. 40:782-790
Kidneys are one of the main dose-limiting organs in radiotherapeutic procedures of lower abdomen. Likewise, the threat of exposure of radiosensitive organs such as kidneys in warfare or radiation accidents among military personal or due to terrorist
Autor:
Bruna Girardi, Maria Pricci, Alfredo Di Leo, Giuseppe Losurdo, Floriana Giorgio, Enzo Ierardi, Andrea Iannone, Michele Barone, Mariabeatrice Principi
Publikováno v:
Carcinogenesis. 39:1274-1282
On the basis of preliminary in vitro experience, we assessed whether an enriched nutritional formulation with estrogen receptor (ER)-beta agonist and anti-inflammatory properties may prevent inflammation-associated colorectal cancer (CRC) in an anima
Autor:
Lee W. Jones, Lauren G. Koch, John N. McGinley, Elizabeth S. Neil, Steven L. Britton, Henry J. Thompson
Publikováno v:
Carcinogenesis. 38:920-928
Although regular physical activity is associated with improvement in aerobic capacity and lower breast cancer risk, there are heritable sets of traits that affect improvement in aerobic capacity in response to physical activity. Although aerobic capa
Autor:
Chiharu Moriya, Hiroaki Taniguchi, Nobuhiro Nishiyama, Kohzoh Imai, Kazunori Kataoka, Kanjiro Miyata
Publikováno v:
Carcinogenesis. 38:638-648
Pancreatic cancer is one of the most lethal types of cancer, with aggressive properties characterized by metastasis, recurrence and drug resistance. Cancer stem cells are considered to be responsible for these properties. PRDM14, a transcriptional re
Autor:
Maria João Gouveia, Lúcio Lara Santos, Thewarach Laha, Maria Y. Pakharukova, Viatcheslav A. Mordvinov, Banchob Sripa, Paul J. Brindley, Gabriel Rinaldi, Nuno Vale, José Manuel Correia da Costa, Teresina Amaro, Galina A. Maksimova
Publikováno v:
Carcinogenesis. 38:929-937
The liver fluke Opisthorchis felineus is a member of the triad of epidemiologically relevant species of the trematode family Opisthorchiidae, and the causative agent of opisthorchiasis felinea over an extensive range that spans regions of Eurasia. Th
Autor:
Melody Baddoo, Chloe Cable, Hani Nakhoul, Suzana D. Savkovic, Erik K. Flemington, Susan E. Crawford, Sandra Heller, Harrison M. Penrose
Publikováno v:
Carcinogenesis. 38:302-311
Obesity, an immense epidemic affecting approximately half a billion adults, has doubled in prevalence in the last several decades. Epidemiological data support that obesity, due to intake of a high-fat, western diet, increases the risk of colon cance
Autor:
Takahiro Kodama, Takuya Inoue, Akira Maekawa, Kenji Watabe, Yoshiki Tsujii, Masahiro Inoue, Tomohide Tatsumi, Yoshito Hayashi, Yasuhiko Tomita, Tomofumi Akasaka, Hayato Hikita, Masahiko Tsujii, Jumpei Kondo, Shinichiro Shinzaki, Tetsuo Takehara, Hideki Iijima, Shunsuke Yoshii
Publikováno v:
Carcinogenesis. 37:972-984
Cancer-associated fibroblasts (CAFs) create a microenvironment that contributes to tumor growth; however, the mechanism by which fibroblasts are phenotypically altered to CAFs remains unclear. Loss or mutation of the tumor suppressor p53 plays a cruc