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Autor:
Hans Walter Lindemann, Peter Staib, Peter Brossart, Donald Bunjes, Claudia Haferlach, R. Kuse, Dieter K. Hossfeld, Nicolaus Kröger, Hartmut Döhner, Rainer Schwerdtfeger, Gabriela M. Baerlocher, Dietrich W. Beelen, Alois Gratwohl, Andreas Hochhaus, Hans-Jochem Kolb, Martin C. Müller, Michele Baccarani, Ruediger Hehlmann, Christian Dietz, Herrad Baumann, Brigitte Schlegelberger, Andreas Reiter, Bernd Hertenstein, Christof Scheid, J Novotny, Michael Lauseker, Lida Kalmanti, Susanne Saussele, Axel R. Zander, Jiri Mayer, Karsten Spiekermann, Susanne Schnittger, Markus Pfirrmann, Joerg Hasford, Axel A. Fauser, Sebastien Rinaldetti, Renate Arnold, Michael Schatz, Matthias Edinger, Martine Jotterand, Arnold Ganser, Herbert G. Sayer, Christoph Nerl, Carlo Aul, Alice Fabarius, Christiane Falge, Matthias Bormann
Publikováno v:
Blood. 126:1581-1581
During the course of chronic myeloid leukemia (CML) progression to blast crisis (BC) is thought to be caused by genetic instability such as cytogenetic aberrations in addition to the translocation t(9;22)(q34;q11). We have shown previously that major