Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Anna Mae Diehl"'
Autor:
Cheng-Shine Hwang, Hui Zhi Lin, S. Costa, Shiqi Yang, Anna Mae Diehl, M D Lane, Helena Cortez-Pinto, G. J. Bagby
Publikováno v:
Biochemical and Biophysical Research Communications. 251:313-319
The liver is a target for bacterial lipopolysaccharide (LPS) and participates in the metabolic response to endotoxemia. Recently published evidence indicates that LPS increases the expression of mitochondrial uncoupling protein-2 (UCP-2) mRNAs in sev
Publikováno v:
Biochemical and biophysical research communications. 350(2)
PPARgamma agonists inhibit liver fibrosis, but the mechanisms involved are uncertain. We hypothesized that PPARgamma agonists inhibit transforming growth factor (TGF)beta1-activation of TGFbeta receptor (TGFbetaR)-1 signaling in quiescent stellate ce
Autor:
Tania Roskams, Nicoletta Sinelli, Jude A. Oben, Michael Torbenson, Zhiping Li, Anna Mae Diehl, Steven A. Thomas, Huizhi Lin, Shiqi Yang
Publikováno v:
Biochemical and biophysical research communications. 308(2)
Leptin’s actions on certain cells require a leptin-inducible neurotransmitter, norepinephrine (NE). NE modulates hepatic fibrosis. Therefore, decreased NE may explain why leptin deficiency inhibits hepatic fibrosis. We manipulated adrenergic activi
Publikováno v:
Biochemical and biophysical research communications. 302(4)
The mechanisms initiating and perpetuating the fibrogenic response in the injured liver are not well understood. Hepatic stellate cells are activated by liver injury to become proliferative and fibrogenic myofibroblasts. Emerging evidence suggests th
Publikováno v:
Biochemical and biophysical research communications. 300(1)
The mechanisms that initiate and perpetuate the fibrogenic response, during liver injury, are unclear. Animal studies, however, strongly support a role for the autonomic nervous system (ANS) in wound healing. Therefore, the ANS may also mediate the d