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Autor:
Rohit Ramchandra, G Clive N May, Marcus Matear, Andrew M. Allen, Daniel T Xing, Gavin W. Lambert
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 317:R386-R396
In heart failure (HF), increases in renal sympathetic nerve activity (RSNA), renal norepinephrine spillover, and renin release cause renal vasoconstriction, which may contribute to the cardiorenal syndrome. To increase our understanding of the mechan
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 312:R903-R911
This study tested the hypothesis that varying degrees of hemodynamic fluctuations seen after birth following immediate cord clamping were related to development of asphyxia with longer cord clamp-to-ventilation intervals, resulting in higher perinata
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 302:R1153-R1157
Patients with congestive heart failure (CHF) due to left ventricular (LV) dysfunction have sympathetic activation specifically directed to the myocardium. Although pulmonary arterial hypertension (PAH) is associated with increased systemic sympatheti
Autor:
Michael Horowitz, Judith M. Wishart, Lora Vanis, Diana Gentilcore, Christopher K. Rayner, Karen L. Jones, Christine Feinle-Bisset
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 300:R1524-R1531
Postprandial hypotension is an important problem, particularly in the elderly. The fall in blood pressure is dependent on small intestinal glucose delivery and, possibly, changes in splanchnic blood flow, the release of glucagon-like peptide-1 (GLP-1
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 289:R23-R28
In humans, multiparity (repeated pregnancy) is associated with increased risk of cardiovascular disease. In rats, multiparity increases the pressor response to phenylephrine and to acute stress, due in part to changes in tone of the splanchnic arteri
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 288:R413-R419
The aim of this study was to determine whether activation of central type II glucocorticoid receptors can blunt autonomic nervous system counterregulatory responses to subsequent hypoglycemia. Sixty conscious unrestrained Sprague-Dawley rats were stu
Autor:
Jennifer L. Bennett, Gerald J. Taborsky, Pam Gronbeck, Charles W. Wilkinson, Dianne P. Figlewicz, Scott B. Evans
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 284:R57-R65
The anatomic connections of the paraventricular nucleus of the hypothalamus (PVN) are such that it is ideally situated to modulate and/or control autonomic responses to a variety of stressors, including hypoglycemia. In our experimental model of hypo
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 283:R1395-R1403
Birth is characterized by a surge in sympathetic outflow, heart rate (HR), mean arterial blood pressure (MABP) and circulating catecholamines. The paraventricular nucleus (PVN) of the hypothalamus is an important central regulatory site of sympatheti
Publikováno v:
Scopus-Elsevier
In response to an acute hypoxemic insult, the mammalian fetus shows a redistribution of the cardiac output in favor of the heart and brain. Peripheral vasoconstriction contributes to this response and is partly mediated by the release of catecholamin
Autor:
Aryana Zavosh, Dianne P. Figlewicz, Pam Gronbeck, Scott B. Evans, Charles W. Wilkinson, Kathy Bentson
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 281:R1426-R1436
The mechanism(s) underlying hypoglycemia-associated autonomic failure (HAAF) are unknown. To test the hypothesis that the activation of brain regions involved in the counterregulatory response to hypoglycemia is blunted with HAAF, rats were studied i