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Autor:
Marlene Thorsen Mørch, Shohreh Issazadeh-Navikas, Thomas Hellesøe Holm, Dina Dræby, Siegfried Weiss, Trevor Owens, Stefan Lienenklaus, Carsten Tue Berg, Ruthe Storgaard Dieu, Reza Khorooshi
Publikováno v:
Acta Neuropathologica
Khorooshi, R, Mørch, M T, Holm, T, Berg, C T, Dieu, R T, Dræby, D, Issazadeh-Navikas, S, Weiss, S, Lienenklaus, S & Owens, T 2015, ' Induction of endogenous Type I interferon within the central nervous system plays a protective role in experimental autoimmune encephalomyelitis ', Acta Neuropathologica, vol. 130, no. 1, pp. 107-118 . https://doi.org/10.1007/s00401-015-1418-z
Khorooshi, R, Mørch, M T, Holm, T, Berg, C T, Dieu, R T, Dræby, D, Issazadeh-Navikas, S, Weiss, S, Lienenklaus, S & Owens, T 2015, ' Induction of endogenous Type I interferon within the central nervous system plays a protective role in experimental autoimmune encephalomyelitis ', Acta Neuropathologica, vol. 130, no. 1, pp. 107-118 . https://doi.org/10.1007/s00401-015-1418-z
The Type I interferons (IFN), beta (IFN-β) and the alpha family (IFN-α), act through a common receptor and have anti-inflammatory effects. IFN-β is used to treat multiple sclerosis (MS) and is effective against experimental autoimmune encephalomye