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Publikováno v:
Thrombosis Research, 207, 143-149. Elsevier Science
Introduction Hormonal contraception is a well-known risk factor for venous thromboembolism (VTE). APC resistance and impaired functions of protein S and TFPI are thought to play an important role in the pathogenesis of hormone-related VTE. It is unkn
Publikováno v:
Thrombosis Research. 198:93-98
Acetylsalicylic acid (ASA) and type 2 diabetes mellitus (T2DM) affect fibrin clot properties through fibrinogen acetylation or glycation. We aimed to identify glycation and acetylation sites on fibrinogen in plasma fibrin clot of T2DM patients with r
Publikováno v:
Thrombosis Research. 196:510-515
Introduction On-treatment platelet aggregability represents the major form of functional assessment for patients treated with P2Y12 receptor antagonists, with “high” on-treatment platelet aggregability (HTPA) predicting thrombotic risk. However H
Autor:
Vibeke Andersen, Corinna Bang, Else-Marie Bladbjerg, Lucas Moitinho-Silva, Claus B. Juhl, Line Espenhain Landgrebe
Publikováno v:
Landgrebe, L E, Juhl, C B, Andersen, V, Moitinho-Silva, L, Bang, C & Bladbjerg, E M 2020, ' Postprandial factor VII activation does not increase plasma concentrations of prothrombin fragment 1 + 2 in patients with morbid obesity ', Thrombosis Research, vol. 196, pp. 260-267 . https://doi.org/10.1016/j.thromres.2020.08.044
Introduction Increased postprandial factor VII activation is observed after high-fat meals, but is not accompanied by thrombin formation in normal weight individuals. Obesity is associated with a higher circulating concentration of tissue factor (TF)
Autor:
Giovanna Marchetti, Annalisa Castagna, Barry Woodhams, Marcello Baroni, Francesco Bernardi, Oliviero Olivieri, Nicola Martinelli, Barbara Lunghi, Mirko Pinotti, Filippo Stefanoni
Publikováno v:
Thrombosis Research. 189:140-146
Background High plasma levels of activated Factor VII-Antithrombin complex (FVIIa-AT) have been associated with an increased risk of cardiovascular mortality in patients with stable coronary artery disease (CAD). Objectives To investigate if FVIIa-AT
Autor:
Zsuzsa Bagoly, Anetta Undas, Agata Hanna Bryk, Elżbieta Broniatowska, Jakub Siudut, Barbara Barath, Éva Katona
Publikováno v:
Thrombosis Research. 185:55-62
Background Type 2 diabetes mellitus (T2DM) is associated with hypofibrinolysis and increased factor XIII-mediated α2-antiplasmin incorporation into the fibrin clot. It is unclear whether there are sex-related differences in α2-antiplasmin incorpora
Autor:
Maria Fernström, Anita Hurtig-Wennlöf, Ulrika Fernberg, Liza U. Ljungberg, Knut Fälker, Madelene Lindkvist, Magnus Grenegård
Publikováno v:
Thrombosis Research. 174:5-12
Introduction Platelet aggregation and secretion can be induced by a large number of endogenous activators, such as collagen, adenosine diphosphate (ADP) and epinephrine. Conversely, the blood vessel endothelium constitutively release platelet inhibit
Autor:
Yuan Long, Yu-Lan Huang, Yongrong Lai, Donghong Deng, Yuling Xu, Peng Cheng, Haimei Liang, Zhenfang Liu, Na Sun
Publikováno v:
Thrombosis Research. 172:61-66
Thalassemia is characterized by a hypercoagulable state in which the protein C (PC) pathway controls thrombosis. We investigated changes in PC, protein S (PS), antithrombin III (AT III) and soluble endothelial protein C receptor (sEPCR) in thalassemi
Protection of the endothelial glycocalyx by antithrombin in an endotoxin-induced rat model of sepsis
Autor:
Tatsuhiko Hirota, Toshiaki Iba, Taisuke Murakami, Makoto Hiki, Jerrold H. Levy, Isao Nagaoka, Koichi Sato
Publikováno v:
Thrombosis Research. 171:1-6
Introduction Injury and loss of the endothelial glycocalyx occur during the early phase of sepsis. We previously showed that antithrombin has a protective effect on this structure in vitro. Here, we investigated the possible protective effects of ant
Autor:
Edna Afonso Reis, Maria da Gloria Carvalho, M.C.J. Guimarães, Júnea Paolucci de Paiva Silvino, Karina Braga Gomes, P.S. Azevedo, Ana Paula Lucas Mota, Maira Cândida Rodrigues de Sousa, Rita Carolina Figueiredo Duarte, Ieda de Fátima Oliveira Silva
Publikováno v:
Thrombosis research. 200
Introduction Familial hypercholesterolemia (FH) is an autosomal dominant genetic disease. The prevalence of FH has previously been reported as 1 in 500 in the general population. This study aimed to evaluate the proprotein convertase subtilisin/kexin